Literature DB >> 18757507

The clinical time-course of experimental autoimmune uveoretinitis using topical endoscopic fundal imaging with histologic and cellular infiltrate correlation.

David A Copland1, Michael S Wertheim, W John Armitage, Lindsay B Nicholson, Ben J E Raveney, Andrew D Dick.   

Abstract

PURPOSE: EAU is an established preclinical model for assessment of immunotherapeutic efficacy toward translation of therapy for posterior uveitis. Reliable screening of clinical features that correlate with underlying retinal changes and damage has not been possible to date. This study was undertaken to describe, validate, and correlate topical endoscopic fundus imaging (TEFI) with histologic features of murine experimental autoimmune uveoretinitis (EAU), with the intent of generating a rapid noninvasive panretinal assessment of ocular inflammation.
METHODS: EAU was induced in B10.RIII mice by immunization with the peptide RBP-3(161-180). The clinical disease course (days 0-63) was monitored and documented using TEFI. Disease severity and pathology were confirmed at various time points by histologic assessment. The composition of the cell infiltrate was also examined and enumerated by flow cytometry.
RESULTS: TEFI demonstrated the hallmark features of EAU, paralleling many of the clinical features of human uveitis, and closely aligned with underlying histologic changes, the severity of which correlated significantly with the number of infiltrating retinal leukocytes. Leukocytic infiltration occurred before manifestation of clinical disease and clinically fulminant disease, as well as cell infiltrate, resolved faster than histologic scores. During the resolution phase, neither the clinical appearance nor number of infiltrating retinal leukocytes returned to predisease levels.
CONCLUSIONS: In EAU, there is a strong correlation between histologic severity and the number of infiltrating leukocytes into the retina. TEFI enhances the monitoring of clinical disease in a rapid and noninvasive fashion. Full assessment of preclinical immunotherapeutic efficacy requires the use of all three parameters: TEFI, histologic assessment, and flow cytometric analysis of retinal infiltrate.

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Year:  2008        PMID: 18757507     DOI: 10.1167/iovs.08-2348

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  34 in total

Review 1.  Doyne lecture 2016: intraocular health and the many faces of inflammation.

Authors:  A D Dick
Journal:  Eye (Lond)       Date:  2016-09-16       Impact factor: 3.775

2.  Tumour necrosis factor-mediated macrophage activation in the target organ is critical for clinical manifestation of uveitis.

Authors:  T K Khera; D A Copland; J Boldison; P J P Lait; D E Szymkowski; A D Dick; L B Nicholson
Journal:  Clin Exp Immunol       Date:  2012-05       Impact factor: 4.330

3.  A portable, contact animal fundus imaging system based on Rol's GRIN lenses.

Authors:  Victor Hernandez; Thomas Albini; William Lee; Cornelis Rowaan; Derek Nankivil; Esdras Arrieta; Jean-Marie Parel
Journal:  Vet Ophthalmol       Date:  2011-08-08       Impact factor: 1.644

4.  Dual Function of the IRF8 Transcription Factor in Autoimmune Uveitis: Loss of IRF8 in T Cells Exacerbates Uveitis, Whereas Irf8 Deletion in the Retina Confers Protection.

Authors:  Sung-Hye Kim; Jenna Burton; Cheng-Rong Yu; Lin Sun; Chang He; Hongsheng Wang; Herbert C Morse; Charles E Egwuagu
Journal:  J Immunol       Date:  2015-07-10       Impact factor: 5.422

5.  TNFR1 signalling is a critical checkpoint for developing macrophages that control of T-cell proliferation.

Authors:  Ben J E Raveney; David A Copland; Claudia J Calder; Andrew D Dick; Lindsay B Nicholson
Journal:  Immunology       Date:  2010-11       Impact factor: 7.397

6.  Systemic and local anti-C5 therapy reduces the disease severity in experimental autoimmune uveoretinitis.

Authors:  D A Copland; K Hussain; S Baalasubramanian; T R Hughes; B P Morgan; H Xu; A D Dick; L B Nicholson
Journal:  Clin Exp Immunol       Date:  2009-12-04       Impact factor: 4.330

7.  A novel pathogenic RBP-3 peptide reveals epitope spreading in persistent experimental autoimmune uveoretinitis.

Authors:  Joanne Boldison; Tarnjit K Khera; David A Copland; Madeleine L Stimpson; Gemma L Crawford; Andrew D Dick; Lindsay B Nicholson
Journal:  Immunology       Date:  2015-08-11       Impact factor: 7.397

Review 8.  Role of the retinal vascular endothelial cell in ocular disease.

Authors:  Arpita S Bharadwaj; Binoy Appukuttan; Phillip A Wilmarth; Yuzhen Pan; Andrew J Stempel; Timothy J Chipps; Eric E Benedetti; David O Zamora; Dongseok Choi; Larry L David; Justine R Smith
Journal:  Prog Retin Eye Res       Date:  2012-09-11       Impact factor: 21.198

9.  SOCS3 deletion in T lymphocytes suppresses development of chronic ocular inflammation via upregulation of CTLA-4 and expansion of regulatory T cells.

Authors:  Cheng-Rong Yu; Sung-Hye Kim; Rashid M Mahdi; Charles E Egwuagu
Journal:  J Immunol       Date:  2013-10-07       Impact factor: 5.422

10.  Oral delivery of ACE2/Ang-(1-7) bioencapsulated in plant cells protects against experimental uveitis and autoimmune uveoretinitis.

Authors:  Pollob K Shil; Kwang-Chul Kwon; Ping Zhu; Amrisha Verma; Henry Daniell; Qiuhong Li
Journal:  Mol Ther       Date:  2014-09-17       Impact factor: 11.454

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