Literature DB >> 18752444

Sickle cell trait is associated with a delayed onset of malaria: implications for time-to-event analysis in clinical studies of malaria.

Peter D Crompton1, Boubacar Traore, Kassoum Kayentao, Safiatou Doumbo, Aissata Ongoiba, Seidina A S Diakite, Michael A Krause, Didier Doumtabe, Younoussou Kone, Greta Weiss, Chiung-Yu Huang, Seydou Doumbia, Aldiouma Guindo, Rick M Fairhurst, Louis H Miller, Susan K Pierce, Ogobara K Doumbo.   

Abstract

BACKGROUND: The World Health Organization (WHO) recently recommended that the time to first malaria episode serve as the primary end point in phase III malaria vaccine trials--the first of which will be held in Africa. Although common red blood cell (RBC) polymorphisms such as sickle hemoglobin (HbS) are known to protect against malaria in Africa, their impact on this end point has not been investigated.
METHODS: A longitudinal study of 225 individuals aged 2-25 years was conducted in Mali. The association between common RBC polymorphisms and the time to first malaria episode was evaluated.
RESULTS: Among children aged 2-10 years, sickle cell trait (HbAS) was associated with a 34-day delay in the median time to first malaria episode (P= .017) Cox regression analysis showed that greater age (hazard ratio [HR], 0.87 [95% CI, 0.80-0.94]; (P= .001), HbAS (HR, 0.48 [95% CI, 0.26-0.91]; (P= .024), and asymptomatic parasitemia at enrollment (HR, 0.35 [95% CI, 0.14-0.85]; (P= .021) were associated with decreased malaria risk.
CONCLUSION: Given the delay in the time to first malaria episode associated with HbAS, it would be advisable for clinical trials and observational studies that use this end point to include Hb typing in the design of studies conducted in areas where HbAS is prevalent.

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Year:  2008        PMID: 18752444      PMCID: PMC2574881          DOI: 10.1086/592224

Source DB:  PubMed          Journal:  J Infect Dis        ISSN: 0022-1899            Impact factor:   5.226


  41 in total

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4.  Sickle cell trait and the risk of Plasmodium falciparum malaria and other childhood diseases.

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10.  An immune basis for malaria protection by the sickle cell trait.

Authors:  Thomas N Williams; Tabitha W Mwangi; David J Roberts; Neal D Alexander; David J Weatherall; Sammy Wambua; Moses Kortok; Robert W Snow; Kevin Marsh
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  70 in total

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4.  A prospective analysis of the Ab response to Plasmodium falciparum before and after a malaria season by protein microarray.

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8.  Humoral and cellular immunity to Plasmodium falciparum merozoite surface protein 1 and protection from infection with blood-stage parasites.

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9.  The Plasmodium falciparum-specific human memory B cell compartment expands gradually with repeated malaria infections.

Authors:  Greta E Weiss; Boubacar Traore; Kassoum Kayentao; Aissata Ongoiba; Safiatou Doumbo; Didier Doumtabe; Younoussou Kone; Seydou Dia; Agnes Guindo; Abdramane Traore; Chiung-Yu Huang; Kazutoyo Miura; Marko Mircetic; Shanping Li; Amy Baughman; David L Narum; Louis H Miller; Ogobara K Doumbo; Susan K Pierce; Peter D Crompton
Journal:  PLoS Pathog       Date:  2010-05-20       Impact factor: 6.823

10.  An intensive longitudinal cohort study of Malian children and adults reveals no evidence of acquired immunity to Plasmodium falciparum infection.

Authors:  Tuan M Tran; Shanping Li; Safiatou Doumbo; Didier Doumtabe; Chiung-Yu Huang; Seydou Dia; Aboudramane Bathily; Jules Sangala; Younoussou Kone; Abdrahamane Traore; Moussa Niangaly; Charles Dara; Kassoum Kayentao; Aissata Ongoiba; Ogobara K Doumbo; Boubacar Traore; Peter D Crompton
Journal:  Clin Infect Dis       Date:  2013-03-13       Impact factor: 9.079

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