Literature DB >> 18692033

Evaluation of lactate as a marker of metabolic stress and cause of secondary damage in acute ischemic stroke or TIA.

Raf Brouns1, Rishi Sheorajpanday, Annick Wauters, Didier De Surgeloose, Peter Mariën, Peter P De Deyn.   

Abstract

BACKGROUND: Accumulation of lactate in ischemic regions has been documented in acute stroke. We evaluated the relation between lactate levels in blood and cerebrospinal fluid (CSF) and ischemic stroke evolution and outcome.
METHODS: Lactate was measured in blood of 187 acute ischemic stroke and TIA patients at admission, 24 h, 72 h and 7 days after stroke onset. In a subpopulation of 85 stroke patients and in 51 controls, lactate was measured in CSF. Stroke evolution was evaluated by change in the NIHSS score within the first 72 h and by occurrence of progressing stroke. At 3 months after stroke, outcome was assessed on the basis of mortality rate and the modified Rankin Scale.
RESULTS: We found no relation between lactate levels in blood and stroke evolution or outcome. Lactate in CSF was higher in stroke patients than in controls and correlated with stroke evolution and outcome. Multivariate regression analyses showed that CSF lactate levels, age and stroke severity are independent predictors for stroke evolution and outcome.
CONCLUSIONS: Lactate levels in CSF, but not in blood, are a reliable marker for metabolic crisis in acute ischemic stroke and correlate with the stroke evolution in the subacute phase and with long-term outcome.

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Year:  2008        PMID: 18692033     DOI: 10.1016/j.cca.2008.07.016

Source DB:  PubMed          Journal:  Clin Chim Acta        ISSN: 0009-8981            Impact factor:   3.786


  20 in total

1.  A 41-year-old man with new headache and altered mental status.

Authors:  Erik C B Johnson; Timothy W West; Nerissa U Ko; Jonathan B Strober
Journal:  Neurohospitalist       Date:  2011-01

2.  Metabolomic Estimation of the Diagnosis and Onset Time of Permanent and Transient Cerebral Ischemia.

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Journal:  Mol Neurobiol       Date:  2017-12-21       Impact factor: 5.590

3.  Prolyl carboxypeptidase activity decline correlates with severity and short-term outcome in acute ischemic stroke.

Authors:  Kaat Kehoe; Raf Brouns; Robert Verkerk; Sebastiaan Engelborghs; Peter Paul De Deyn; Dirk Hendriks; Ingrid De Meester
Journal:  Neurochem Res       Date:  2014-11-05       Impact factor: 3.996

4.  The role of tryptophan catabolism along the kynurenine pathway in acute ischemic stroke.

Authors:  Raf Brouns; Robert Verkerk; Tony Aerts; Didier De Surgeloose; Annick Wauters; Simon Scharpé; Peter P De Deyn
Journal:  Neurochem Res       Date:  2010-05-19       Impact factor: 3.996

5.  Dimethylarginine levels in cerebrospinal fluid of hyperacute ischemic stroke patients are associated with stroke severity.

Authors:  Raf Brouns; Bart Marescau; Ilse Possemiers; Rishi Sheorajpanday; Peter P De Deyn
Journal:  Neurochem Res       Date:  2009-03-19       Impact factor: 3.996

6.  Acute Ischemic Stroke Severity, Progression, and Outcome Relate to Changes in Dipeptidyl Peptidase IV and Fibroblast Activation Protein Activity.

Authors:  Lesley Baerts; Raf Brouns; Kaat Kehoe; Robert Verkerk; Sebastiaan Engelborghs; Peter Paul De Deyn; Dirk Hendriks; Ingrid De Meester
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Review 7.  Phytochemicals in Ischemic Stroke.

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8.  Hunting mimics and chameleons: diagnostic difficulties in atypical acute ischaemic stroke.

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Journal:  BMJ Case Rep       Date:  2014-06-02

Review 9.  Advanced monitoring of systemic hemodynamics in critically ill patients with acute brain injury.

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Journal:  Neurocrit Care       Date:  2014-12       Impact factor: 3.210

Review 10.  Metabolic Reprogramming: Strategy for Ischemic Stroke Treatment by Ischemic Preconditioning.

Authors:  Jing Liang; Rongrong Han; Bing Zhou
Journal:  Biology (Basel)       Date:  2021-05-11
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