Literature DB >> 18640936

DNA damage and repair capacity in patients with lung cancer: prediction of multiple primary tumors.

Irene Orlow1, Bernard J Park, Urvi Mujumdar, Himali Patel, Puiki Siu-Lau, Brian A Clas, Robert Downey, Raja Flores, Manjit Bains, Nabil Rizk, Gemma Dominguez, Jen Jani, Marianne Berwick, Colin B Begg, Mark G Kris, Valerie W Rusch.   

Abstract

PURPOSE: Patients who survive one occurrence of non-small-cell lung cancer (NSCLC) are at higher risk of a second malignancy. Capacity to repair damaged DNA may modulate individual susceptibility to develop lung cancer. Therefore, we evaluated constitutive and induced DNA damage, and repair capacity, in patients with multiple NSCLCs (cases) and compared the results to those obtained in patients with a single NSCLC (controls). PATIENTS AND METHODS: One hundred eight cases and 99 controls matched by age, sex, and time since diagnosis were studied. DNA damage was assessed on peripheral blood lymphocytes by the comet assay before and after exposing cells to a tobacco-derived carcinogen, using the tail moment and the tail intensity as measures to assess baseline damage, induced damage and repair capacity.
RESULTS: Constitutive DNA damage, benzo(a)pyrene diol epoxide-induced damage, and repair after BPDE-induced damage were all significantly higher in cases than in controls. These results were confirmed in regression analyses adjusted for potential confounders.
CONCLUSION: DNA damage as measured by the comet assay is associated with the development of multiple primary tumors in individuals with NSCLC.

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Year:  2008        PMID: 18640936      PMCID: PMC4423737          DOI: 10.1200/JCO.2007.13.2654

Source DB:  PubMed          Journal:  J Clin Oncol        ISSN: 0732-183X            Impact factor:   44.544


  41 in total

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Authors:  Q Wei; J Gu; L Cheng; M L Bondy; H Jiang; W K Hong; M R Spitz
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6.  Detection of DNA damage in peripheral blood mononuclear cells from pancreatic cancer patients.

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