Literature DB >> 18633125

Influence of peptidylarginine deiminase type 4 genotype and shared epitope on clinical characteristics and autoantibody profile of rheumatoid arthritis.

B Hoppe1, T Häupl, K Egerer, R Gruber, H Kiesewetter, A Salama, G R Burmester, T Dörner.   

Abstract

BACKGROUND: Recent evidence suggests that distinction of subsets of rheumatoid arthritis (RA) depending on anti-cyclic citrullinated peptide antibody (anti-CCP) status may be helpful in distinguishing distinct aetiopathologies and in predicting the course of disease. HLA-DRB1 shared epitope (SE) and peptidylarginine deiminase type 4 (PADI4) genotype, both of which have been implicated in anti-CCP generation, are assumed to be associated with RA.
OBJECTIVES: To elucidate whether PADI4 affects the clinical characteristics of RA, and whether it would modulate the effect of anti-CCPs on clinical course. The combined effect of SE and PADI4 on autoantibody profile was also analysed.
METHODS: 373 patients with RA were studied. SE, padi4_94C>T, rheumatoid factor, anti-CCPs and antinuclear antibodies (ANAs) were determined. Disease severity was characterised by cumulative therapy intensity classified into ordinal categories (CTI-1 to CTI-3) and by Steinbrocker score.
RESULTS: CTI was significantly associated with disease duration, erosive disease, disease activity score (DAS) 28 and anti-CCPs. The association of anti-CCPs with CTI was considerably influenced by padi4_94C>T genotype (C/C: OR(adj) = 0.93, p(adj) = 0.92; C/T: OR(adj) = 2.92, p(adj) = 0.093; T/T: OR(adj) = 15.3, p(adj) = 0.002). Carriage of padi4_94T exhibited a significant trend towards higher Steinbrocker scores in univariate and multivariate analyses. An association of padi4_94C>T with ANAs was observed, with noteworthy differences depending on SE status (SE-: OR(adj) = 6.20, p(adj)<0.04; SE+: OR(adj) = 0.36, p(adj) = 0.02) and significant heterogeneity between the two SE strata (p = 0.006).
CONCLUSIONS: PADI4 genotype in combination with anti-CCPs and SE modulates clinical and serological characteristics of RA.

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Year:  2008        PMID: 18633125     DOI: 10.1136/ard.2008.091983

Source DB:  PubMed          Journal:  Ann Rheum Dis        ISSN: 0003-4967            Impact factor:   19.103


  11 in total

1.  [Biomarkers in rheumatology. Biomarkers and imaging for the diagnosis and stratification of rheumatoid arthritis and spondylarthritis in the ArthroMark network funded by the Federal Ministry of Education and Research].

Authors:  T Häupl; H Appel; M Backhaus; H Burkhardt; M Grünke; A Grützkau; B Hoppe; J Listing; B Ostendorf; M Rudwaleit; J Sieper; A Skapenko; K Skriner; T Sörensen; B Stuhlmüller; A Zink; H Schulze-Koops; G-R Burmester
Journal:  Z Rheumatol       Date:  2012-06       Impact factor: 1.372

2.  Citrullination of NF-κB p65 promotes its nuclear localization and TLR-induced expression of IL-1β and TNFα.

Authors:  Bo Sun; Nishant Dwivedi; Tyler J Bechtel; Janet L Paulsen; Aaron Muth; Mandar Bawadekar; Gang Li; Paul R Thompson; Miriam A Shelef; Celia A Schiffer; Eranthie Weerapana; I-Cheng Ho
Journal:  Sci Immunol       Date:  2017-06-09

3.  A molecular signature of preclinical rheumatoid arthritis triggered by dysregulated PTPN22.

Authors:  Hui-Hsin Chang; Guang-Yaw Liu; Nishant Dwivedi; Bo Sun; Yuko Okamoto; Jennifer D Kinslow; Kevin D Deane; M Kristen Demoruelle; Jill M Norris; Paul R Thompson; Jeffrey A Sparks; Deepak A Rao; Elizabeth W Karlson; Hui-Chih Hung; V Michael Holers; I-Cheng Ho
Journal:  JCI Insight       Date:  2016-10-20

4.  Peptidyl arginine deiminase type IV (PADI4) haplotypes interact with shared epitope regardless of anti-cyclic citrullinated peptide antibody or erosive joint status in rheumatoid arthritis: a case control study.

Authors:  So-Young Bang; Tae-Un Han; Chan-Bum Choi; Yoon-Kyoung Sung; Sang-Cheol Bae; Changwon Kang
Journal:  Arthritis Res Ther       Date:  2010-06-10       Impact factor: 5.156

5.  Polymorphisms in peptidylarginine deiminase associate with rheumatoid arthritis in diverse Asian populations: evidence from MyEIRA study and meta-analysis.

Authors:  Chun Lai Too; Shahnaz Murad; Jasbir Singh Dhaliwal; Per Larsson; Xia Jiang; Bo Ding; Lars Alfredsson; Lars Klareskog; Leonid Padyukov
Journal:  Arthritis Res Ther       Date:  2012-11-19       Impact factor: 5.156

6.  PADI2 is significantly associated with rheumatoid arthritis.

Authors:  Xiaotian Chang; Yifang Xia; Jihong Pan; Qingsong Meng; Yan Zhao; Xinfeng Yan
Journal:  PLoS One       Date:  2013-12-05       Impact factor: 3.240

7.  PADI4 genotype is not associated with rheumatoid arthritis in a large UK Caucasian population.

Authors:  Marian L Burr; Haris Naseem; Anne Hinks; Steve Eyre; Laura J Gibbons; John Bowes; Anthony G Wilson; James Maxwell; Ann W Morgan; Paul Emery; Sophia Steer; Lynne Hocking; David M Reid; Paul Wordsworth; Pille Harrison; Wendy Thomson; Jane Worthington; Anne Barton
Journal:  Ann Rheum Dis       Date:  2009-05-25       Impact factor: 19.103

8.  PADI4 and HLA-DRB1 are genetic risks for radiographic progression in RA patients, independent of ACPA status: results from the IORRA cohort study.

Authors:  Taku Suzuki; Katsunori Ikari; Koichiro Yano; Eisuke Inoue; Yoshiaki Toyama; Atsuo Taniguchi; Hisashi Yamanaka; Shigeki Momohara
Journal:  PLoS One       Date:  2013-04-08       Impact factor: 3.240

9.  Pharmacogenetics of rheumatoid arthritis: Potential targets from susceptibility genes and present therapies.

Authors:  Darren D O'Rielly; Proton Rahman
Journal:  Pharmgenomics Pers Med       Date:  2010-03-30

10.  Hypogalactosylation of immunoglobulin G in rheumatoid arthritis: relationship to HLA-DRB1 shared epitope, anticitrullinated protein antibodies, rheumatoid factor, and correlation with inflammatory activity.

Authors:  Christian Schwedler; Thomas Häupl; Ulrich Kalus; Véronique Blanchard; Gerd-Rüdiger Burmester; Denis Poddubnyy; Berthold Hoppe
Journal:  Arthritis Res Ther       Date:  2018-03-14       Impact factor: 5.156

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