Literature DB >> 18597064

[Postoperative manifestation of acute compartment syndrome by previously unknown heterozygote sickle cell anemia. A clinical case report].

A Schulz1, A C Disch, I Melcher, N P Haas, K-D Schaser.   

Abstract

Patients suffering from sickle cell disease show an increased ischemic intolerance due to continuous pro-inflammatory activation and dysfunction of the endothelium by recurrent vaso-occlusive episodes. The presented case shows the manifestation of a postoperative compartment syndrome of the lower leg following the intraoperative use of blood arrest in a patient with previously unknown sickle cell disease. Preexisting vulnerability for tissue hypoxia in patients at risk should be a major concern for intraoperative use of blood arrest during surgery of the extremities.

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Year:  2008        PMID: 18597064     DOI: 10.1007/s00113-008-1446-4

Source DB:  PubMed          Journal:  Unfallchirurg        ISSN: 0177-5537            Impact factor:   1.000


  16 in total

1.  Rate of deoxygenation modulates rheologic behavior of sickle red blood cells at a given mean corpuscular hemoglobin concentration.

Authors:  D K Kaul; X D Liu
Journal:  Clin Hemorheol Microcirc       Date:  1999       Impact factor: 2.375

2.  Hypoxia/reoxygenation causes inflammatory response in transgenic sickle mice but not in normal mice.

Authors:  D K Kaul; R P Hebbel
Journal:  J Clin Invest       Date:  2000-08       Impact factor: 14.808

Review 3.  The perfusion paradox and vascular instability in sickle cell disease.

Authors:  Karl A Nath; Zvonimir S Katusic; Mark T Gladwin
Journal:  Microcirculation       Date:  2004-03       Impact factor: 2.628

4.  Oxygen radical inhibition of nitric oxide-dependent vascular function in sickle cell disease.

Authors:  M Aslan; T M Ryan; B Adler; T M Townes; D A Parks; J A Thompson; A Tousson; M T Gladwin; R P Patel; M M Tarpey; I Batinic-Haberle; C R White; B A Freeman
Journal:  Proc Natl Acad Sci U S A       Date:  2001-12-18       Impact factor: 11.205

5.  Myonecrosis and myofibrosis as complications of sickle cell anemia.

Authors:  J Valeriano-Marcet; L D Kerr
Journal:  Ann Intern Med       Date:  1991-07-15       Impact factor: 25.391

Review 6.  Pathophysiology and related studies of the no reflow phenomenon in skeletal muscle.

Authors:  D M Allen; L E Chen; A V Seaber; J R Urbaniak
Journal:  Clin Orthop Relat Res       Date:  1995-05       Impact factor: 4.176

Review 7.  Sickle cell anemia as a rheologic disease.

Authors:  M K Horne
Journal:  Am J Med       Date:  1981-02       Impact factor: 4.965

8.  Reperfusion injury pathophysiology in sickle transgenic mice.

Authors:  U R Osarogiagbon; S Choong; J D Belcher; G M Vercellotti; M S Paller; R P Hebbel
Journal:  Blood       Date:  2000-07-01       Impact factor: 22.113

Review 9.  Sickle myonecrosis revisited.

Authors:  S Mani; T P Duffy
Journal:  Am J Med       Date:  1993-11       Impact factor: 4.965

Review 10.  Reperfusion injury.

Authors:  Alizan A Khalil; Farah A Aziz; John C Hall
Journal:  Plast Reconstr Surg       Date:  2006-03       Impact factor: 4.730

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