Literature DB >> 18591451

Mechanisms of cardiac dysfunction associated with tyrosine kinase inhibitor cancer therapeutics.

Ming Hui Chen1, Risto Kerkelä, Thomas Force.   

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Year:  2008        PMID： 18591451      PMCID： PMC2735334          DOI： 10.1161/CIRCULATIONAHA.108.776831

Source DB:  PubMed          Journal:  Circulation        ISSN： 0009-7322            Impact factor:   29.690


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  75 in total

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5.  FGF1/p38 MAP kinase inhibitor therapy induces cardiomyocyte mitosis, reduces scarring, and rescues function after myocardial infarction.

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6.  BNP as a marker of the heart failure in the treatment of imatinib mesylate.

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8.  Safety, pharmacokinetic, and antitumor activity of SU11248, a novel oral multitarget tyrosine kinase inhibitor, in patients with cancer.

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10.  Activation of AMP-activated protein kinase by human EGF receptor 2/EGF receptor tyrosine kinase inhibitor protects cardiac cells.

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1.  Use of Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes (hiPSC-CMs) to Monitor Compound Effects on Cardiac Myocyte Signaling Pathways.

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2.  Recurrent myocardial infarction associated with gefitinib therapy.

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Review 5.  Heart Failure in Relation to Tumor-Targeted Therapies and Immunotherapies.

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7.  Efficacy and learning curve of a hand-held echocardiography device in an oncology outpatient clinic: Expanding the use of echoscopic heart examination beyond cardiology.

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8.  Aortic stiffness increases upon receipt of anthracycline chemotherapy.

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9.  Critical role of nuclear calcium/calmodulin-dependent protein kinase IIdeltaB in cardiomyocyte survival in cardiomyopathy.

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10.  Identification of a kinase profile that predicts chromosome damage induced by small molecule kinase inhibitors.

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Journal:  PLoS Comput Biol       Date:  2009-07-24       Impact factor: 4.475
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