Literature DB >> 18588867

Elevated expression of IL-17 and IL-12 genes in chronic inflammatory periodontal disease.

Tomoyuki Honda1, Yukari Aoki, Naoki Takahashi, Tomoki Maekawa, Takako Nakajima, Harue Ito, Koichi Tabeta, Takafumi Okui, Keiko Kajita, Hisanori Domon, Kazuhisa Yamazaki.   

Abstract

BACKGROUND: A number of different theories have been postulated to explain the progression of gingivitis to periodontitis in the context of the Th1/Th2 paradigm. However, no consistent results have been obtained. Th17, a new T-cell subset producing IL-17, which is implicated in many aspect of inflammatory tissue destruction, overcomes many of the discrepant findings in the studies related to the Th1/Th2 hypothesis. We compared the gene expression profile of Th17-related molecules in gingivitis and periodontitis lesions showing distinct clinical entities.
METHODS: Gingival tissue samples were obtained from 23 gingivitis and 24 periodontitis tissues. The gene expression was measured by using quantitative real-time PCR for IL-17A, IL-17F, CCR4, CCR6, IL-12 p35 and IL-23 p19. The difference of gene expressions between gingivitis and periodontitis was analyzed by Mann-Whitney U-test. Correlations between each gene expression were also analyzed.
RESULTS: The expression level of IL-17A was higher than that of IL-17F and a significant difference in expression between gingivitis and periodontitis was observed only for IL-17A. CCR4 and CCR6 tended to be higher in periodontitis compared with gingivitis, although the differences were not statistically significant. Whereas the gene expression of IL-12 p35 was significantly higher in periodontitis compared with gingivitis, that of IL-23 p19 was not different between the two diseases.
CONCLUSION: This study demonstrates the elevated expression of IL-17 and IL-12 in periodontitis, i.e., the tissue destruction form of periodontal diseases, as compared with gingivitis, and provides new insight into the T-cell mediated immunopathogenesis of periodontal disease.

Entities:  

Mesh:

Substances:

Year:  2008        PMID: 18588867     DOI: 10.1016/j.cca.2008.06.003

Source DB:  PubMed          Journal:  Clin Chim Acta        ISSN: 0009-8981            Impact factor:   3.786


  20 in total

1.  Clinical associations between IL-17 family cytokines and periodontitis and potential differential roles for IL-17A and IL-17E in periodontal immunity.

Authors:  Raja Azman Awang; Raja Azman; David F Lappin; Alexandrea MacPherson; Marcello Riggio; Douglas Robertson; Penny Hodge; Gordon Ramage; Shauna Culshaw; Philip M Preshaw; John Taylor; Christopher Nile
Journal:  Inflamm Res       Date:  2014-11-05       Impact factor: 4.575

2.  TLR2 signaling and Th2 responses drive Tannerella forsythia-induced periodontal bone loss.

Authors:  Srinivas R Myneni; Rajendra P Settem; Terry D Connell; Achsah D Keegan; Sarah L Gaffen; Ashu Sharma
Journal:  J Immunol       Date:  2011-06-01       Impact factor: 5.422

3.  Alveolar Bone Protective Effect of Hiziki Extracts on the Progression of Periodontitis.

Authors:  Don-Gil Lee; Yu-Kyong Shin; Jae-Hee Park; Sang-Yong Park; Eunson Hwang; Jung-Eun Yang; Hae Jo; Ki-Young Kim; Gafurjon T Mavlonov; Tae-Hoo Yi
Journal:  Mar Biotechnol (NY)       Date:  2018-04-14       Impact factor: 3.619

4.  Systemic Antibiotic Therapy Reduces Circulating Inflammatory Dendritic Cells and Treg-Th17 Plasticity in Periodontitis.

Authors:  Mythilypriya Rajendran; Stephen Looney; Nagendra Singh; Mahmoud Elashiry; Mohamed M Meghil; Ahmed R El-Awady; Omnia Tawfik; Cristiano Susin; Roger M Arce; Christopher W Cutler
Journal:  J Immunol       Date:  2019-04-03       Impact factor: 5.422

Review 5.  Restoring host-microbe homeostasis via selective chemoattraction of Tregs.

Authors:  G P Garlet; C S Sfeir; S R Little
Journal:  J Dent Res       Date:  2014-07-23       Impact factor: 6.116

Review 6.  IL-17: overview and role in oral immunity and microbiome.

Authors:  L Abusleme; N M Moutsopoulos
Journal:  Oral Dis       Date:  2016-12-27       Impact factor: 3.511

7.  IL-17 in sera from patients with aggressive periodontitis.

Authors:  H A Schenkein; T E Koertge; C N Brooks; R Sabatini; D E Purkall; J G Tew
Journal:  J Dent Res       Date:  2010-04-16       Impact factor: 6.116

8.  Protease-activated receptor 2 has pivotal roles in cellular mechanisms involved in experimental periodontitis.

Authors:  David M Wong; Vivian Tam; Roselind Lam; Katrina A Walsh; Liliana Tatarczuch; Charles N Pagel; Eric C Reynolds; Neil M O'Brien-Simpson; Eleanor J Mackie; Robert N Pike
Journal:  Infect Immun       Date:  2009-11-23       Impact factor: 3.441

Review 9.  Basic biology and role of interleukin-17 in immunity and inflammation.

Authors:  Camille Zenobia; George Hajishengallis
Journal:  Periodontol 2000       Date:  2015-10       Impact factor: 7.589

10.  Osteoimmunological mechanisms involved in orthodontically and bacterially induced periodontal stress.

Authors:  A Konermann; W Götz; D Wohlleber; P Knolle; J Deschner; A Jäger
Journal:  J Orofac Orthop       Date:  2012-12       Impact factor: 1.938

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.