Literature DB >> 18581058

The smoker's paradox after successful fibrinolysis: reduced risk of reocclusion but no improved long-term cardiac outcome.

Peter C Kievit1, Marc A Brouwer, Gerrit Veen, Wim R M Aengevaeren, Freek W A Verheugt.   

Abstract

BACKGROUND: In smokers treated with fibrinolysis for ST-elevation myocardial infarction (STEMI) a paradoxical beneficial short-term outcome has been reported. This was attributed to favorable clinical and angiographic baseline variables and a better response to fibrinolysis. During follow-up infarct artery reocclusion is an important prognosticator. We studied the effects of smoking on reocclusion and long-term cardiac outcome after successful fibrinolysis.
METHODS: In the Antithrombotics in the Prevention of Reocclusion In COronary Thrombolysis trials (APRICOT-1 and -2) 499 STEMI patients with an open infarct artery <48 h after fibrinolysis received randomized antithrombotic treatment until 3-month follow-up angiography. Five-year clinical follow-up was complete.
RESULTS: Current smokers (317 patients, 64%) had favorable clinical (age 54 vs. 60 years, P < 0.01) and angiographic (single vessel disease 61% vs. 49%, P = 0.02) baseline characteristics. Reocclusion rates were 21% (67/317) in smokers versus 32% (59/182) in non-smokers (P < 0.01). Five-year infarct-free cardiac survival did not differ: 82% vs. 85%. Reocclusion (HR 2.41, 95%CI 1.05-5.56, P = 0.04) independently predicted cardiac mortality. Smoking was independently associated with a reduced risk of reocclusion (OR 0.58, 95%CI 0.37-0.91, P = 0.02), but not with improved 5-year cardiac outcome (HR 1.34, 95%CI 0.79-2.25, P = ns).
CONCLUSIONS: After successful fibrinolysis, smoking is independently associated with a more than 40% reduced risk of reocclusion, which is an independent predictor of adverse outcome. However, even with more favorable baseline characteristics smokers did not have improved 5-year cardiac outcome in this low-risk population.

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Year:  2008        PMID: 18581058     DOI: 10.1007/s11239-008-0238-6

Source DB:  PubMed          Journal:  J Thromb Thrombolysis        ISSN: 0929-5305            Impact factor:   2.300


  36 in total

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2.  Consequences of reocclusion after successful reperfusion therapy in acute myocardial infarction. TAMI Study Group.

Authors:  E M Ohman; R M Califf; E J Topol; R Candela; C Abbottsmith; S Ellis; K N Sigmon; D Kereiakes; B George; R Stack
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Review 3.  Update on the role of cigarette smoking in coronary artery disease.

Authors:  W B Kannel
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4.  The Thrombolysis in Myocardial Infarction (TIMI) trial. Phase I findings.

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5.  Reperfusion therapy for acute myocardial infarction with fibrinolytic therapy or combination reduced fibrinolytic therapy and platelet glycoprotein IIb/IIIa inhibition: the GUSTO V randomised trial.

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7.  Aspirin plus coumarin versus aspirin alone in the prevention of reocclusion after fibrinolysis for acute myocardial infarction: results of the Antithrombotics in the Prevention of Reocclusion In Coronary Thrombolysis (APRICOT)-2 Trial.

Authors:  Marc A Brouwer; Paul J P C van den Bergh; Wim R M Aengevaeren; Gerrit Veen; Hans E Luijten; Don P Hertzberger; Ad J van Boven; Ralf P J W Vromans; Gérard J H Uijen; Freek W A Verheugt
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8.  Effect of cigarette smoking on outcome after thrombolytic therapy for myocardial infarction.

Authors:  C L Grines; E J Topol; W W O'Neill; B S George; D Kereiakes; H R Phillips; J D Leimberger; L H Woodlief; R M Califf
Journal:  Circulation       Date:  1995-01-15       Impact factor: 29.690

9.  The effects of tissue plasminogen activator, streptokinase, or both on coronary-artery patency, ventricular function, and survival after acute myocardial infarction.

Authors: 
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Authors:  E J Topol; R M Califf; B S George; D J Kereiakes; C W Abbottsmith; R J Candela; K L Lee; B Pitt; R S Stack; W W O'Neill
Journal:  N Engl J Med       Date:  1987-09-03       Impact factor: 91.245

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