Literature DB >> 18565333

Ovalbumin-protein sigma 1 M-cell targeting facilitates oral tolerance with reduction of antigen-specific CD4+ T cells.

Hideaki Suzuki1, Shinichi Sekine, Kosuke Kataoka, David W Pascual, Massimo Maddaloni, Ryoki Kobayashi, Keiko Fujihashi, Haruo Kozono, Jerry R McGhee, Kohtaro Fujihashi.   

Abstract

BACKGROUND & AIMS: The follicle-associated epithelium (FAE) plays key roles in antigen uptake and subsequent induction of mucosal immunity. In this study, we examined whether M-cell targeting using a protein antigen (Ag) delivery system would induce oral tolerance instead of enhancement of Ag-specific mucosal antibody (Ab) responses.
METHODS: Mice were fed different doses of a recombinant protein sigma 1 of reovirus genetically conjugated to ovalbumin (OVA-psigma1), psigma1 only, or phosphate-buffered saline (PBS) before oral challenge with OVA plus cholera toxin as mucosal adjuvant. OVA-specific Ab and CD4-positive (CD4(+)) T-cell responses were determined.
RESULTS: A low dose of OVA-psigma1 reduced anti-OVA Ab and CD4(+) T-cell responses in both mucosal and systemic lymphoid tissues. OVA/MHC I-A(d) tetramer staining showed that the numbers of OVA-specific CD4(+) T cells were significantly reduced in lamina propria of mice fed OVA-psigma1 than those fed psigma1 only or PBS only. In fact, Foxp3 expressing CD25(+) CD4(+) T cells were markedly increased in this tissue. Nonetheless, CD25(+) CD4(+) T cells from the spleen, mesenteric lymph nodes, and Peyer's patches of orally tolerized mice showed increased transforming growth factor beta1 (TGF-beta1) and interleukin-10 (IL-10) production compared with nontolerized mice.
CONCLUSIONS: These results show that an FAE M-cell targeting protein Ag delivery system facilitates oral tolerance induction because of a reduction in Ag-specific CD4(+) T cells and increased levels of TGF-beta1 and IL-10 producing, CD25(+) CD4(+) regulatory T cells in both systemic and mucosal lymphoid tissues.

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Year:  2008        PMID: 18565333      PMCID: PMC2579966          DOI: 10.1053/j.gastro.2008.05.037

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   22.682


  40 in total

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Authors:  Y Wu; M J Boysun; K L Csencsits; D W Pascual
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2.  Oral tolerance induction with antigen conjugated to cholera toxin B subunit generates both Foxp3+CD25+ and Foxp3-CD25- CD4+ regulatory T cells.

Authors:  Jia-Bin Sun; Sukanya Raghavan; Asa Sjöling; Samuel Lundin; Jan Holmgren
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3.  Induction of rapid T cell activation and tolerance by systemic presentation of an orally administered antigen.

Authors:  I Gütgemann; A M Fahrer; J D Altman; M M Davis; Y H Chien
Journal:  Immunity       Date:  1998-06       Impact factor: 31.745

Review 4.  Oral tolerance in disease.

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Review 5.  Oral tolerance.

Authors:  W Strober; B Kelsall; T Marth
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6.  Oral tolerance revisited: prior oral tolerization abrogates cholera toxin-induced mucosal IgA responses.

Authors:  H Kato; K Fujihashi; R Kato; Y Yuki; J R McGhee
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Review 7.  Oral tolerance in the treatment of inflammatory autoimmune diseases.

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9.  Expanding dendritic cells in vivo enhances the induction of oral tolerance.

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Review 6.  Oral tolerance.

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10.  IL-28 supplants requirement for T(reg) cells in protein sigma1-mediated protection against murine experimental autoimmune encephalomyelitis (EAE).

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