Literature DB >> 18443776

Endovascular treatment in proximal and intracranial carotid occlusion 9 hours after symptom onset.

Malgorzata M Jakubowska1, Peter Michels, Axel Müller-Jensen, Andreas Leppien, Bernd Eckert.   

Abstract

INTRODUCTION: A debate is emerging over whether the treatment time window in acute stroke can be extended beyond 6 h if penumbral tissue can be identified. Treatment decisions are very difficult in cases of tandem proximal carotid occlusion with arterioarterial intracranial embolism. We enter this debate with the present report on a case of atherosclerotic proximal carotid occlusion and resulting periocclusional carotid T embolism that was successfully treated 9 h after symptom onset.
METHODS: The case of a 68-year-old man with fluctuating symptoms of right-hemispheric stroke is presented (NIHSS score 12-20 on admission). CT angiography demonstrated proximal carotid occlusion and periocclusional embolism of the entire internal carotid artery (ICA) including the carotid T segment. Penumbral tissue was diagnosed by nonenhanced and perfusion CT imaging 7.5 h after symptom onset. Treatment was initiated 9 h after symptom onset by passing the proximal occlusion with a microcatheter and local administration of recombinant tissue plasminogen activator (rt-PA) into the carotid T segment at the level of posterior communicating artery (PCoA) origin.
RESULTS: Recanalization of the ICA and middle cerebral artery was accomplished within 1 h by flow establishment via the PCoA. The atherosclerotic proximal ICA occlusion was not stented due to the risk of embolism from remnant thrombi in the petrous and cavernous ICA segments. Follow-up MRI showed only mild haemorrhagic infarct transformation of the initial infarct core. The patient was discharged from hospital 18 days after treatment with NIHSS score 5.
CONCLUSION: If penumbral tissue can be conclusively identified, endovascular treatment in proximal and intracranial tandem occlusion can be successful, even in treatments initiated 6-9 h after stroke onset. If the intracranial flow after recanalization can be established via the circle of Willis, the underlying proximal ICA occlusion may not require treatment.

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Year:  2008        PMID: 18443776     DOI: 10.1007/s00234-008-0385-7

Source DB:  PubMed          Journal:  Neuroradiology        ISSN: 0028-3940            Impact factor:   2.804


  11 in total

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7.  Tissue plasminogen activator for acute ischemic stroke.

Authors: 
Journal:  N Engl J Med       Date:  1995-12-14       Impact factor: 91.245

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9.  Thrombolytic therapy in acute occlusion of the intracranial internal carotid artery bifurcation.

Authors:  O Jansen; R von Kummer; M Forsting; W Hacke; K Sartor
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10.  Effect of intravenous thrombolysis on MRI parameters and functional outcome in acute stroke <6 hours.

Authors:  J Röther; P D Schellinger; A Gass; M Siebler; A Villringer; J B Fiebach; J Fiehler; O Jansen; T Kucinski; V Schoder; K Szabo; G J Junge-Hülsing; M Hennerici; H Zeumer; K Sartor; C Weiller; W Hacke
Journal:  Stroke       Date:  2002-10       Impact factor: 7.914

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2.  Potential for the use of the Solitaire stent for recanalization of middle cerebral artery occlusion without a susceptibility vessel sign.

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