Literature DB >> 18440223

Defects in insulin signaling pathways in ovarian steroidogenesis and other tissues in polycystic ovary syndrome (PCOS).

Evanthia Diamanti-Kandarakis1, Georgia Argyrakopoulou, Frangiskos Economou, Eleni Kandaraki, Michael Koutsilieris.   

Abstract

The polycystic ovary syndrome (PCOS) is the most common endocrinopathy in women of reproductive age today. Women with PCOS often demonstrate defective ovarian steroid biosynthesis and present with hyperandrogenemia. Moreover, 50-70% of PCOS women are insulin resistant and hyperinsulinemic. Insulin acts on the ovary via its own receptor and interacts with gonadotrophins, modulating steroidogenesis. The precise role of insulin and the molecular mechanisms that take place are not yet completely explicated. This review will be focused on insulin's action on the ovary and other target tissues, describing the intracellular signaling pathways implicated in steroidogenesis and their defects in women with PCOS.

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Year:  2008        PMID: 18440223     DOI: 10.1016/j.jsbmb.2008.03.014

Source DB:  PubMed          Journal:  J Steroid Biochem Mol Biol        ISSN: 0960-0760            Impact factor:   4.292


  31 in total

Review 1.  Conserved insulin signaling in the regulation of oocyte growth, development, and maturation.

Authors:  Debabrata Das; Swathi Arur
Journal:  Mol Reprod Dev       Date:  2017-04-24       Impact factor: 2.609

2.  Developmental programming: effect of prenatal steroid excess on intraovarian components of insulin signaling pathway and related proteins in sheep.

Authors:  Hugo H Ortega; Florencia Rey; Melisa M L Velazquez; Vasantha Padmanabhan
Journal:  Biol Reprod       Date:  2010-02-10       Impact factor: 4.285

Review 3.  Scientific Statement on the Diagnostic Criteria, Epidemiology, Pathophysiology, and Molecular Genetics of Polycystic Ovary Syndrome.

Authors:  Daniel A Dumesic; Sharon E Oberfield; Elisabet Stener-Victorin; John C Marshall; Joop S Laven; Richard S Legro
Journal:  Endocr Rev       Date:  2015-10       Impact factor: 19.871

Review 4.  Animal Models to Understand the Etiology and Pathophysiology of Polycystic Ovary Syndrome.

Authors:  Elisabet Stener-Victorin; Vasantha Padmanabhan; Kirsty A Walters; Rebecca E Campbell; Anna Benrick; Paolo Giacobini; Daniel A Dumesic; David H Abbott
Journal:  Endocr Rev       Date:  2020-07-01       Impact factor: 19.871

5.  Insulin signaling displayed a differential tissue-specific response to low-dose dihydrotestosterone in female mice.

Authors:  Stanley Andrisse; Katelyn Billings; Ping Xue; Sheng Wu
Journal:  Am J Physiol Endocrinol Metab       Date:  2017-12-19       Impact factor: 4.310

6.  Dietary underreporting in women affected by polycystic ovary syndrome: A pilot study.

Authors:  Rachele De Giuseppe; Valentina Braschi; David Bosoni; Ginevra Biino; Fatima C Stanford; Rossella E Nappi; Hellas Cena
Journal:  Nutr Diet       Date:  2018-08-05       Impact factor: 2.333

7.  Interactions between metabolic and reproductive functions in the resumption of postpartum fecundity.

Authors:  Claudia Valeggia; Peter T Ellison
Journal:  Am J Hum Biol       Date:  2009 Jul-Aug       Impact factor: 1.937

8.  Polycystic ovary syndrome is associated with tissue-specific differences in insulin resistance.

Authors:  Theodore P Ciaraldi; Vanita Aroda; Sunder Mudaliar; R Jeffrey Chang; Robert R Henry
Journal:  J Clin Endocrinol Metab       Date:  2008-10-14       Impact factor: 5.958

Review 9.  Skeletal muscle insulin resistance in endocrine disease.

Authors:  Melpomeni Peppa; Chrysi Koliaki; Panagiotis Nikolopoulos; Sotirios A Raptis
Journal:  J Biomed Biotechnol       Date:  2010-03-15

10.  Overexpression of Lnk in the Ovaries Is Involved in Insulin Resistance in Women With Polycystic Ovary Syndrome.

Authors:  Meihua Hao; Feng Yuan; Chenchen Jin; Zehong Zhou; Qi Cao; Ling Xu; Guanlei Wang; Hui Huang; Dongzi Yang; Meiqing Xie; Xiaomiao Zhao
Journal:  Endocrinology       Date:  2016-07-26       Impact factor: 4.736

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