Literature DB >> 18434216

Absence of association of asporin polymorphisms and osteoarthritis susceptibility in US Caucasians.

U Atif1, A Philip, J Aponte, E M Woldu, S Brady, V B Kraus, J M Jordan, M Doherty, A G Wilson, R W Moskowitz, M Hochberg, R Loeser, J B Renner, M Chiano.   

Abstract

OBJECTIVE: An association between osteoarthritis (OA) and functional polymorphisms in the aspartic acid (d) repeat of the asporin (ASPN) gene was reported in Japanese and Han Chinese populations. The aim of this study was to assess the association of variants in the ASPN gene with the presence of radiographic hand and/or knee OA in a US Caucasian population.
METHODS: Ten single nucleotide polymorphisms (SNPs) within the ASPN gene were genotyped in 775 affected siblings with radiographically confirmed hand and/or knee OA, and the allelic, genotypic and haplotypic association results were examined.
RESULTS: One variant (SNP RS7033979) showed nominal evidence of association with both hand OA (P=0.042) and knee OA (P=0.032). Four additional SNPs showed nominal evidence of association with knee OA only. These associations were only observed with genotypic tests; the corresponding allelic and haplotype tests did not corroborate the single-point association results.
CONCLUSION: These data suggest that polymorphisms within ASPN are not a major influence in susceptibility to hand or knee OA in US Caucasians.

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Year:  2008        PMID: 18434216      PMCID: PMC3664276          DOI: 10.1016/j.joca.2008.03.007

Source DB:  PubMed          Journal:  Osteoarthritis Cartilage        ISSN: 1063-4584            Impact factor:   6.576


  17 in total

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Authors:  V B Kraus; J M Jordan; M Doherty; A G Wilson; R Moskowitz; M Hochberg; R Loeser; M Hooper; J B Renner; M M Crane; P Hastie; S Sundseth; U Atif
Journal:  Osteoarthritis Cartilage       Date:  2006-11-17       Impact factor: 6.576

2.  Replication of the association of the aspartic acid repeat polymorphism in the asporin gene with knee-osteoarthritis susceptibility in Han Chinese.

Authors:  Qing Jiang; Dongquan Shi; Long Yi; Shiro Ikegawa; Yong Wang; Takahiro Nakamura; Di Qiao; Cheng Liu; Jin Dai
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3.  Expression pattern and gene characterization of asporin. a newly discovered member of the leucine-rich repeat protein family.

Authors:  S P Henry; M Takanosu; T C Boyd; P M Mayne; H Eberspaecher; W Zhou; B de Crombrugghe; M Hook; R Mayne
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4.  Sex and ethnic differences in the association of ASPN, CALM1, COL2A1, COMP, and FRZB with genetic susceptibility to osteoarthritis of the knee.

Authors:  Ana M Valdes; John Loughlin; Mark Van Oene; Kay Chapman; Gabriela L Surdulescu; Michael Doherty; Tim D Spector
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7.  An aspartic acid repeat polymorphism in asporin inhibits chondrogenesis and increases susceptibility to osteoarthritis.

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9.  Association of the aspartic acid-repeat polymorphism in the asporin gene with age at onset of knee osteoarthritis in Han Chinese population.

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  15 in total

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Authors:  Linda J Sandell
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Journal:  Clin Cancer Res       Date:  2015-10-07       Impact factor: 12.531

4.  A meta-analysis of the relationship between aspartic acid (D)-repeat polymorphisms in asporin and osteoarthritis susceptibility.

Authors:  Gwan Gyu Song; Jae-Hoon Kim; Young Ho Lee
Journal:  Rheumatol Int       Date:  2013-12-05       Impact factor: 2.631

5.  Association of the D repeat polymorphism in the ASPN gene with developmental dysplasia of the hip: a case-control study in Han Chinese.

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7.  Functional polymorphisms in asporin and CILP together with joint loading predispose to hand osteoarthritis.

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8.  Association of aspartic acid repeat polymorphism in the asporin gene with osteoarthritis of knee, hip, and hand: A PRISMA-compliant meta-analysis.

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9.  Genetic polymorphism in the asporin gene is not a key risk factor for osteoarthritis: Evidence based on an updated cumulative meta-analysis.

Authors:  Jing Wang; Aimin Yang; Jie Zhang; Na Sun; Xiangwen Li; Xinghui Li; Qiling Liu; Jun Li; Xiaomei Ren; Zunhua Ke; Rongqiang Zhang
Journal:  Exp Ther Med       Date:  2018-02-26       Impact factor: 2.447

Review 10.  Developments in the scientific understanding of osteoarthritis.

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