Literature DB >> 18362597

Alteration of the piglet diaphragm contractility in vivo and its recovery after acute hypercapnia.

Samir Jaber1, Boris Jung, Mustapha Sebbane, Michèle Ramonatxo, Xavier Capdevila, Jacques Mercier, Jean-Jacques Eledjam, Stefan Matecki.   

Abstract

BACKGROUND: The effects of hypercapnic acidosis on the diaphragm and its recovery to normocapnia have been poorly evaluated. The authors studied diaphragmatic contractility facing acute variations of arterial carbon dioxide tension (Paco2) and evaluated the contractile function at 60 min after normocapnia recovery.
METHODS: Thirteen piglets weighing 15-20 kg were anesthetized, ventilated, and separated into two groups: a control group (n = 5) evaluated in normocapnia (time-control experiments) and a hypercapnia group (n = 8) in which animals were acutely and shortly exposed to five consecutive ranges of Paco2 (40, 50, 70, 90, and 110 mmHg). Then carbon dioxide insufflation was stopped. Diaphragmatic contractility was assessed by measuring transdiaphragmatic pressure variations obtained after bilateral transjugular phrenic nerve pacing at increased frequencies (20-120 Hz). For each level of arterial pressure of carbon dioxide, pressure-frequency curves were obtained in vivo by phrenic nerve pacing.
RESULTS: In the hypercapnia group, mean +/- SD transdiaphragmatic pressure significantly decreased from 41 +/- 3 to 29 +/- 3 cm H2O (P < 0.05) between the first (40 mmHg) and fifth (116 mmHg) stages of capnia at the frequency of 100 Hz stimulation. The observed alteration of the contractile force was proportional to the level of Paco2 (r = 0.61, P < 0.01). Normocapnia recuperation allowed a partial recovery of the diaphragmatic contractile force (80% of the baseline value) at 60 min after carbon dioxide insufflation interruption.
CONCLUSION: A short exposure to respiratory acidosis decreased diaphragmatic contractility proportionally to the degree of hypercapnia, and this alteration was only partially reversed at 60 min after exposure.

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Year:  2008        PMID: 18362597      PMCID: PMC2789322          DOI: 10.1097/ALN.0b013e31816725a6

Source DB:  PubMed          Journal:  Anesthesiology        ISSN: 0003-3022            Impact factor:   7.892


  42 in total

1.  Cardiac output measurement by arterial thermodilution in piglets.

Authors:  G Marx; R Sümpelmann; T Schuerholz; E Thorns; J Heine; B Vangerow; H Rueckoldt
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2.  Effects of prolonged mechanical ventilation and inactivity on piglet diaphragm function.

Authors:  Peter J Radell; Sten Remahl; David G Nichols; Lars I Eriksson
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3.  The effect of sedative drugs on diaphragmatic contractility in dogs: propofol versus midazolam.

Authors:  Y Fujii; T Hoshi; S Takahashi; H Toyooka
Journal:  Anesth Analg       Date:  2000-10       Impact factor: 5.108

Review 4.  Permissive hypercapnia.

Authors:  Keith G Hickling
Journal:  Respir Care Clin N Am       Date:  2002-06

5.  Effects of prolonged mechanical ventilation on respiratory muscle ultrastructure and mitochondrial respiration in rabbits.

Authors:  Nathalie Bernard; Stefan Matecki; Guillaume Py; Sandrine Lopez; Jacques Mercier; Xavier Capdevila
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6.  Cerebral blood flow and metabolism during and after prolonged hypercapnia in newborn lambs.

Authors:  J K Hino; B L Short; K Rais-Bahrami; W R Seale
Journal:  Crit Care Med       Date:  2000-10       Impact factor: 7.598

7.  Effect of hypercapnia on maximal voluntary ventilation and diaphragm fatigue in normal humans.

Authors:  G F Rafferty; M Lou Harris; M I Polkey; A Greenough; J Moxham
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Authors:  V J Cardenas; J B Zwischenberger; W Tao; P D Nguyen; T Schroeder; L D Traber; D L Traber; A Bidani
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9.  Contribution of respiratory acidosis to diaphragmatic fatigue at exercise.

Authors:  S Jonville; N Delpech; A Denjean
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10.  Effects of controlled mechanical ventilation on respiratory muscle contractile properties in rabbits.

Authors:  Xavier Capdevila; Sandrine Lopez; Nathalie Bernard; Emmanuel Rabischong; Michèle Ramonatxo; Guilhem Martinazzo; Christian Prefaut
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  12 in total

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Review 2.  [Ventilator-induced diaphragm dysfunction : clinically relevant problem].

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4.  Intraoperative end-tidal carbon dioxide concentrations: what is the target?

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5.  Hypercapnia attenuates ventilator-induced diaphragm atrophy and modulates dysfunction.

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Journal:  Crit Care       Date:  2014-02-09       Impact factor: 9.097

6.  Clinical signs, profound acidemia, hypoglycemia, and hypernatremia are predictive of mortality in 1,400 critically ill neonatal calves with diarrhea.

Authors:  Florian M Trefz; Ingrid Lorenz; Annette Lorch; Peter D Constable
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7.  Prolonged mechanical ventilation worsens sepsis-induced diaphragmatic dysfunction in the rat.

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Journal:  PLoS One       Date:  2018-08-01       Impact factor: 3.240

Review 8.  Critical illness-associated diaphragm weakness.

Authors:  Martin Dres; Ewan C Goligher; Leo M A Heunks; Laurent J Brochard
Journal:  Intensive Care Med       Date:  2017-09-15       Impact factor: 17.440

9.  Recovery of diaphragm function following mechanical ventilation in a rodent model.

Authors:  Christian S Bruells; Ingmar Bergs; Rolf Rossaint; Jun Du; Christian Bleilevens; Andreas Goetzenich; Joachim Weis; Michael P Wiggs; Scott K Powers; Marc Hein
Journal:  PLoS One       Date:  2014-01-27       Impact factor: 3.240

10.  Moderate and prolonged hypercapnic acidosis may protect against ventilator-induced diaphragmatic dysfunction in healthy piglet: an in vivo study.

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Journal:  Crit Care       Date:  2013-01-24       Impact factor: 9.097

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