Literature DB >> 18314228

Novel PSEN1 and PGRN mutations in early-onset familial frontotemporal dementia.

Livia Bernardi1, Carmine Tomaino, Maria Anfossi, Maura Gallo, Silvana Geracitano, Angela Costanzo, Rosanna Colao, Gianfranco Puccio, Francesca Frangipane, Sabrina A M Curcio, Maria Mirabelli, Nicoletta Smirne, David Iapaolo, Raffaele Giovanni Maletta, Amalia C Bruni.   

Abstract

BACKGROUND: Frontotemporal dementia is a clinically and genetically heterogeneous syndrome. Mutations in two genes, Microtubule Associated Protein Tau (MAPT) and Progranulin (PGRN), and rarely Presenilin mutations, have been causally linked to this disorder.
OBJECTIVE: To investigate the presence of PGRN, PSEN1, PSEN2 and APP mutations in a group of familial early-onset frontotemporal dementia (f-EOFTD) patients negative for MAPT gene mutations. SUBJECTS AND METHODS: We prospectively studied 17 unrelated subjects diagnosed with f-EOFTD (one case neuropathologically confirmed as FTD-Ub+). Among these subjects eight belonged to eight autosomal dominant families unrelated to each other, and nine had at least one first degree relative affected by dementia.
RESULTS: We identified two novel heterozygous mutations in two unrelated patients, Cys139Arg in the PGRN gene and Val412Ile in the PSEN1 gene.
CONCLUSIONS: Early-onset f-FTD remains a heterogeneous disorder from a genetic point of view. PGRN mutation frequency was low in our sample. The presence of a novel PSEN1 mutation suggests that presenilin molecular studies should be performed when screening for MAPT and PGRN genes is negative.

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Year:  2008        PMID: 18314228     DOI: 10.1016/j.neurobiolaging.2008.01.005

Source DB:  PubMed          Journal:  Neurobiol Aging        ISSN: 0197-4580            Impact factor:   4.673


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