Literature DB >> 18311749

Effects of postmenopausal hormone therapy on rheumatoid arthritis: the women's health initiative randomized controlled trials.

Brian Walitt1, Mary Pettinger, Arthur Weinstein, James Katz, James Torner, Mary Chester Wasko, Barbara V Howard.   

Abstract

OBJECTIVE: To study the effects of postmenopausal hormone therapy (PHT) on the incidence and severity of rheumatoid arthritis (RA).
METHODS: The Women's Health Initiative randomized controlled trials evaluated the effects of unopposed estrogen (E-alone) and estrogen plus progestin (E+P) compared with placebo on a diverse set of health outcomes over 7.1 and 5.6 years, respectively. RA cases were identified using historical and medication data. The hazard of developing RA was estimated using Cox proportional hazards regression models. Disease symptom severity was estimated using the Short Form 36 (SF-36) and self-reported joint pain scores at baseline and after 1 year. Mean changes in severity were compared using linear regression models.
RESULTS: Of the 27,347 participants, 63 prevalent cases and 105 incident cases of RA were identified. A nonsignificant reduction in the risk of developing RA (hazard ratio 0.74; 95% confidence interval [95% CI] 0.51, 1.10) was noted with PHT use. PHT use led to improved SF-36 scores in unadjusted analyses (percent change 12.5%; 95% CI -24.45, -0.57) but not after adjustment for relevant covariates (P = 0.33). Nonsignificant improvements in joint pain scores were seen with PHT use (odds ratio [OR] 4.10; 95% CI 0.83, 20.20). PHT did not improve swelling (OR 1.27; 95% CI 0.08, 19.63) or prevent new joint pains (OR 0.72; 95% CI 0.11, 4.68) in RA participants.
CONCLUSION: There were no statistically significant differences in the risk of developing RA or the severity of RA between the PHT and placebo groups.

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Year:  2008        PMID: 18311749      PMCID: PMC2661110          DOI: 10.1002/art.23325

Source DB:  PubMed          Journal:  Arthritis Rheum        ISSN: 0004-3591


  38 in total

1.  Case-control study of rheumatoid arthritis and prior use of oral contraceptives.

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Authors:  S Mathur; R S Mathur; J M Goust; H O Williamson; H H Fudenberg
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3.  Reduction in incidence of rheumatoid arthritis associated with oral contraceptives. Royal College of General Practitioners' Oral Contraception Study.

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4.  Effects of conjugated equine estrogen in postmenopausal women with hysterectomy: the Women's Health Initiative randomized controlled trial.

Authors:  Garnet L Anderson; Marian Limacher; Annlouise R Assaf; Tamsen Bassford; Shirley A A Beresford; Henry Black; Denise Bonds; Robert Brunner; Robert Brzyski; Bette Caan; Rowan Chlebowski; David Curb; Margery Gass; Jennifer Hays; Gerardo Heiss; Susan Hendrix; Barbara V Howard; Judith Hsia; Allan Hubbell; Rebecca Jackson; Karen C Johnson; Howard Judd; Jane Morley Kotchen; Lewis Kuller; Andrea Z LaCroix; Dorothy Lane; Robert D Langer; Norman Lasser; Cora E Lewis; JoAnn Manson; Karen Margolis; Judith Ockene; Mary Jo O'Sullivan; Lawrence Phillips; Ross L Prentice; Cheryl Ritenbaugh; John Robbins; Jacques E Rossouw; Gloria Sarto; Marcia L Stefanick; Linda Van Horn; Jean Wactawski-Wende; Robert Wallace; Sylvia Wassertheil-Smoller
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5.  Oral contraceptives and rheumatoid arthritis: further evidence for a preventive effect.

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6.  Analysis of thymocyte subpopulations following treatment with sex hormones.

Authors:  E A Novotny; E S Raveche; S Sharrow; M Ottinger; A D Steinberg
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7.  17 beta-estradiol regulates cytokine release through modulation of CD16 expression in monocytes and monocyte-derived macrophages.

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8.  The incidence and severity of rheumatoid arthritis, results from a county register in Oslo, Norway.

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9.  Estrogen and other female reproductive risk factors are not strongly associated with the development of rheumatoid arthritis in elderly women.

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Review 2.  Modulation of autoimmune rheumatic diseases by oestrogen and progesterone.

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Review 3.  Sexual dimorphism of RA manifestations: genes, hormones and behavior.

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6.  Health-related quality of life in midlife women in Qatar: relation to arthritis and symptoms of joint pain.

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7.  Association of environmental and genetic factors and gene-environment interactions with risk of developing rheumatoid arthritis.

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9.  A Prospective Study Investigating Prediagnostic Leukocyte Telomere Length and Risk of Developing Rheumatoid Arthritis in Women.

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Review 10.  Why are women predisposed to autoimmune rheumatic diseases?

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