Literature DB >> 18195268

The hypocretin neurotransmission system in myotonic dystrophy type 1.

E Ciafaloni1, E Mignot, V Sansone, J E Hilbert, L Lin, X Lin, L C Liu, W R Pigeon, M L Perlis, C A Thornton.   

Abstract

BACKGROUND: Patients with myotonic dystrophy type 1 (DM1) frequently have symptoms of excessive daytime sleepiness (EDS). Some patients with DM1 show sleep-onset REM, similar to that observed in narcolepsy. Narcolepsy is characterized by impaired hypocretin (Hcrt) neurotransmission.
OBJECTIVE: To test for dysregulation of Hcrt neurotransmission in a prospective cohort of patients with DM1.
METHODS: Hcrt levels in CSF were measured by radioimmunoassay. Sleep physiology was assessed by overnight polysomnography (PSG) and a multiple sleep latency test (MSLT). Splicing of Hcrt receptor 1 and 2 (HcrtR1 and HcrtR2) mRNA was examined in postmortem samples of temporal cortex.
RESULTS: Seventeen of 38 patients with DM1 reported symptoms of EDS. Among patients with DM1 with EDS who underwent PSG/MSLT, 7 of 13 showed reduced sleep latency, sleep-onset REM, or both. However, CSF Hcrt levels in DM1 (mean 277 pg/mL, n = 38) were not different from controls (mean 277 pg/mL, n = 33). Also, splicing of HcrtR1 and HcrtR2 mRNA in patients with DM1 was similar to controls.
CONCLUSIONS: Excessive daytime sleepiness and dysregulation of REM sleep occur frequently in patients with myotonic dystrophy type 1 (DM1). However, the pathophysiologic basis is distinct from narcolepsy, as patients with DM1 do not have a consistent defect of Hcrt release or receptor splicing.

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Year:  2008        PMID: 18195268     DOI: 10.1212/01.wnl.0000296827.20167.98

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  19 in total

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Review 4.  Myotonic dystrophy.

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10.  Grey and white matter loss along cerebral midline structures in myotonic dystrophy type 2.

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Journal:  J Neurol       Date:  2008-09-03       Impact factor: 4.849

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