Literature DB >> 18089693

Global adiposity rather than abnormal regional fat distribution characterizes women with polycystic ovary syndrome.

Thomas M Barber1, Stephen J Golding, Christopher Alvey, John A H Wass, Fredrik Karpe, Stephen Franks, Mark I McCarthy.   

Abstract

CONTEXT: Obesity-related predisposition to polycystic ovary syndrome (PCOS) could reflect overall adiposity and/or regional accumulation of abdominal visceral fat.
OBJECTIVE: The objective of the study was to compare distributions of visceral, abdominal sc, and gluteofemoral sc adipose tissue in PCOS cases vs. control women.
DESIGN: This was a cross-sectional study. SETTING AND PARTICIPANTS: Fat depot measurements from axial magnetic resonance imaging scans taken at anatomically predefined sites were compared between 22 body mass index (BMI)/fat mass-matched pairs of PCOS cases and controls; whole-group comparisons included 50 PCOS cases vs. 28 female controls. All subjects were of UK British/Irish origin. MAIN OUTCOME MEASURE(S): We measured cross-sectional areas of adipose tissue within visceral (mid-L4), abdominal (mid-L4) sc, and gluteofemoral (greater trochanteric and midfemoral) sc fat depots. Other measurements included fat mass, BMI, testosterone, SHBG, and homeostasis model assessment of insulin resistance (a measure of insulin sensitivity). Whole-group analyses were adjusted for fat mass and age.
RESULTS: There were no significant differences in fat-depot measurements between BMI/fat mass-matched pairs of PCOS cases and controls: mid-L4 visceral (P=0.40), abdominal sc (P=0.22), gluteal sc (P=0.67), and midfemoral sc (P=0.37) depots. Whole-group comparisons gave similar results after adjustments for fat mass and age. Fasting serum insulin concentrations (P=0.03) and homeostasis model assessment of insulin resistance (P=0.03) were significantly higher in the PCOS group than BMI/fat mass-matched controls.
CONCLUSIONS: PCOS cases and BMI/fat mass-matched control women are indistinguishable with respect to distribution of fat within visceral, abdominal sc, and gluteofemoral sc depots, despite significant differences in insulin resistance between these two groups.

Entities:  

Mesh:

Year:  2007        PMID: 18089693     DOI: 10.1210/jc.2007-2117

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  49 in total

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3.  Metabolic Evidence of Diminished Lipid Oxidation in Women With Polycystic Ovary Syndrome.

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Journal:  Curr Metabolomics       Date:  2014

4.  Biochemical hyperandrogenism is associated with metabolic syndrome independently of adiposity and insulin resistance in Romanian polycystic ovary syndrome patients.

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10.  Developmental programming: impact of prenatal testosterone excess on insulin sensitivity, adiposity, and free fatty acid profile in postpubertal female sheep.

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Journal:  Endocrinology       Date:  2013-03-24       Impact factor: 4.736

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