Literature DB >> 18079693

Frequency of CEP290 c.2991_1655A>G mutation in 175 Spanish families affected with Leber congenital amaurosis and early-onset retinitis pigmentosa.

Elena Vallespin1, Miguel-Angel Lopez-Martinez, Diego Cantalapiedra, Rosa Riveiro-Alvarez, Jana Aguirre-Lamban, Almudena Avila-Fernandez, Cristina Villaverde, Maria-Jose Trujillo-Tiebas, Carmen Ayuso.   

Abstract

PURPOSE: Leber congenital amaurosis (LCA) is the most severe inherited retinopathy with the earliest age of onset. To date, eleven genes have been reported to cause the non-syndromic LCA phenotype. The CEP290 gene has been shown to account for Joubert and Senior-Loken syndromes and to represent a frequent cause of non-syndromic LCA. The aim of the present study was to establish the prevalence of CEP290 c.2991_1655A>G in non-syndromic Spanish patients having LCA or early-onset retinitis pigmentosa (RP).
METHODS: We used automated sequencing to examine 49 non-syndromic Spanish families with LCA and 126 Spanish families with early-onset RP for the CEP290 c.2991_1655A>G mutation. As a control, we recruited 50 unrelated Spanish healthy individuals.
RESULTS: The frequencies of mutated alleles were 6% in LCA cases and 0% in early-onset RP and healthy individual controls. These results were compared to other populations.
CONCLUSIONS: The CEP290 c.2991_1655A>G mutation frequency in Spanish non-syndromic LCA families is lower than that of other countries.

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Year:  2007        PMID: 18079693

Source DB:  PubMed          Journal:  Mol Vis        ISSN: 1090-0535            Impact factor:   2.367


  9 in total

1.  In vitro and in vivo rescue of aberrant splicing in CEP290-associated LCA by antisense oligonucleotide delivery.

Authors:  Alejandro Garanto; Daniel C Chung; Lonneke Duijkers; Julio C Corral-Serrano; Muriël Messchaert; Ru Xiao; Jean Bennett; Luk H Vandenberghe; Rob W J Collin
Journal:  Hum Mol Genet       Date:  2016-04-22       Impact factor: 6.150

2.  CRISPR/Cas9-Mediated Genome Editing as a Therapeutic Approach for Leber Congenital Amaurosis 10.

Authors:  Guo-Xiang Ruan; Elizabeth Barry; Dan Yu; Michael Lukason; Seng H Cheng; Abraham Scaria
Journal:  Mol Ther       Date:  2017-01-18       Impact factor: 11.454

3.  Mutations that are a common cause of Leber congenital amaurosis in northern America are rare in southern India.

Authors:  Periasamy Sundaresan; P Vijayalakshmi; Stewart Thompson; Audrey C Ko; John H Fingert; Edwin M Stone
Journal:  Mol Vis       Date:  2009-09-04       Impact factor: 2.367

4.  Genetic screening of LCA in Belgium: predominance of CEP290 and identification of potential modifier alleles in AHI1 of CEP290-related phenotypes.

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5.  Exome capture sequencing identifies a novel mutation in BBS4.

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Review 6.  From the cytoplasm into the cilium: bon voyage.

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7.  Antisense Oligonucleotide-Based Splicing Correction in Individuals with Leber Congenital Amaurosis due to Compound Heterozygosity for the c.2991+1655A>G Mutation in CEP290.

Authors:  Lonneke Duijkers; L Ingeborgh van den Born; John Neidhardt; Nathalie M Bax; Laurence H M Pierrache; B Jeroen Klevering; Rob W J Collin; Alejandro Garanto
Journal:  Int J Mol Sci       Date:  2018-03-07       Impact factor: 5.923

8.  Noncoding mutation in RPGRIP1 contributes to inherited retinal degenerations.

Authors:  Gang Zou; Tao Zhang; Xuesen Cheng; Austin D Igelman; Jun Wang; Xinye Qian; Shangyi Fu; Keqing Wang; Robert K Koenekoop; Gerald A Fishman; Paul Yang; Yumei Li; Mark E Pennesi; Rui Chen
Journal:  Mol Vis       Date:  2021-03-18       Impact factor: 2.367

9.  A Mild Phenotype Caused by Two Novel Compound Heterozygous Mutations in CEP290.

Authors:  Agnieszka Rafalska; Anna M Tracewska; Anna Turno-Kręcicka; Milena J Szafraniec; Marta Misiuk-Hojło
Journal:  Genes (Basel)       Date:  2020-10-22       Impact factor: 4.096

  9 in total

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