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5 in total

1. Termination of cardiac Ca(2+) sparks: an investigative mathematical model of calcium-induced calcium release.

Authors: Eric A Sobie; Keith W Dilly; Jader dos Santos Cruz; W Jonathan Lederer; M Saleet Jafri
Journal: Biophys J Date: 2002-07 Impact factor: 4.033

2. The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium.

Authors: Inna Györke; Nichole Hester; Larry R Jones; Sandor Györke
Journal: Biophys J Date: 2004-04 Impact factor: 4.033

3. Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane.

Authors: L Zhang; J Kelley; G Schmeisser; Y M Kobayashi; L R Jones
Journal: J Biol Chem Date: 1997-09-12 Impact factor: 5.157

4. Triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytes.

Authors: Dmitry Terentyev; Steven E Cala; Timothy D Houle; Serge Viatchenko-Karpinski; Inna Gyorke; Radmila Terentyeva; Simon C Williams; Sandor Gyorke
Journal: Circ Res Date: 2005-02-24 Impact factor: 17.367

5. Protein protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes.

Authors: Dmitry Terentyev; Serge Viatchenko-Karpinski; Srikanth Vedamoorthyrao; Sridhar Oduru; Inna Györke; Simon C Williams; Sandor Györke
Journal: J Physiol Date: 2007-06-14 Impact factor: 5.182

5 in total