Literature DB >> 18039947

Severe neonatal manifestations of Costello syndrome.

I F M Lo, C Brewer, N Shannon, J Shorto, B Tang, G Black, M T Soo, D K K Ng, S T S Lam, B Kerr.   

Abstract

BACKGROUND: Costello syndrome (CS) is due to mutations in HRAS, with the most common mutation being c.34G>A (p.G12S), found in most patients in all the published series. A small number of less common mutations have been reported. POPULATION STUDIED: HRAS mutation analysis has been undertaken in 74 predominantly British patients with a possible diagnosis of CS. A HRAS mutation was found in 27 patients, 15 of whom have been previously reported. PHENOTYPE ANALYSIS: Four cases had an unusually severe phenotype, associated in three cases with two unusual mutations, c.35G>A, p.G12D in two cases and c.34G>T, p.G12C in the other. Hypoglycaemia, renal abnormalities, severe early cardiomyopathy, congenital lung and airway abnormalities, pleural and pericardial effusion, chylous ascites and pulmonary lymphangectasia are confirmed as part of the clinical spectrum seen in CS. A lung pathology resembling alveolar capillary dysplasia is reported in one case.
CONCLUSION: These cases illustrate that the diagnosis of CS may be difficult in the newborn period, and should be considered in the differential diagnosis of the sick newborn infant with multisystem disease. Study of more cases will be required to establish if there is a definite association between severe disease and less common mutations.

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Year:  2007        PMID: 18039947     DOI: 10.1136/jmg.2007.054411

Source DB:  PubMed          Journal:  J Med Genet        ISSN: 0022-2593            Impact factor:   6.318


  20 in total

1.  Costello syndrome: Clinical phenotype, genotype, and management guidelines.

Authors:  Karen W Gripp; Lindsey A Morse; Marni Axelrad; Kathryn C Chatfield; Aaron Chidekel; William Dobyns; Daniel Doyle; Bronwyn Kerr; Angela E Lin; David D Schwartz; Barbara J Sibbles; Dawn Siegel; Suma P Shankar; David A Stevenson; Mihir M Thacker; K Nicole Weaver; Sue M White; Katherine A Rauen
Journal:  Am J Med Genet A       Date:  2019-06-20       Impact factor: 2.802

2.  Prenatal features of Costello syndrome: ultrasonographic findings and atrial tachycardia.

Authors:  Angela E Lin; Barbara O'Brien; Laurie A Demmer; Kristina K Almeda; Cynthia L Blanco; Patrick F Glasow; Charles I Berul; Robert Hamilton; A Micheil Innes; Julie L Lauzon; Katia Sol-Church; Karen W Gripp
Journal:  Prenat Diagn       Date:  2009-07       Impact factor: 3.050

3.  Respiratory system involvement in Costello syndrome.

Authors:  Natalia Gomez-Ospina; Christin Kuo; Amitha Lakshmi Ananth; Angela Myers; Marie-Luise Brennan; David A Stevenson; Jonathan A Bernstein; Louanne Hudgins
Journal:  Am J Med Genet A       Date:  2016-04-22       Impact factor: 2.802

4.  A novel HRAS substitution (c.266C>G; p.S89C) resulting in decreased downstream signaling suggests a new dimension of RAS pathway dysregulation in human development.

Authors:  Karen W Gripp; Eugenia Bifeld; Deborah L Stabley; Elizabeth Hopkins; Stefanie Meien; Kathy Vinette; Katia Sol-Church; Georg Rosenberger
Journal:  Am J Med Genet A       Date:  2012-07-20       Impact factor: 2.802

5.  Hyperinsulinemic Hypoglycemia in a Patient with Costello Syndrome: An Etiology to Consider in Hypoglycemia.

Authors:  Dogus Vuralli; Can Kosukcu; Ekim Taskiran; Pelin Ozlem Simsek-Kiper; Gulen Eda Utine; Koray Boduroglu; Ayfer Alikasifoglu; Mehmet Alikasifoglu
Journal:  Mol Syndromol       Date:  2020-09-16

6.  Early-lethal Costello syndrome due to rare HRAS Tandem Base substitution (c.35_36GC>AA; p.G12E)-associated pulmonary vascular disease.

Authors:  K Nicole Weaver; Dehua Wang; James Cnota; Nicholas Gardner; Deborah Stabley; Katia Sol-Church; Karen W Gripp; David P Witte; Kevin E Bove; Robert J Hopkin
Journal:  Pediatr Dev Pathol       Date:  2014-08-18

7.  An attenuated phenotype of Costello syndrome in three unrelated individuals with a HRAS c.179G>A (p.Gly60Asp) mutation correlates with uncommon functional consequences.

Authors:  Karen W Gripp; Katia Sol-Church; Patroula Smpokou; Gail E Graham; David A Stevenson; Heather Hanson; David H Viskochil; Laura C Baker; Bridget Russo; Nick Gardner; Deborah L Stabley; Verena Kolbe; Georg Rosenberger
Journal:  Am J Med Genet A       Date:  2015-04-25       Impact factor: 2.802

8.  Pathogenic variant in EPHB4 results in central conducting lymphatic anomaly.

Authors:  Dong Li; Tara L Wenger; Christoph Seiler; Michael E March; Alvaro Gutierrez-Uzquiza; Charlly Kao; Elizabeth Bhoj; Lifeng Tian; Misha Rosenbach; Yichuan Liu; Nora Robinson; Mechenzie Behr; Rosetta Chiavacci; Cuiping Hou; Tiancheng Wang; Marina Bakay; Renata Pellegrino da Silva; Jonathan A Perkins; Patrick Sleiman; Michael A Levine; Patricia J Hicks; Maxim Itkin; Yoav Dori; Hakon Hakonarson
Journal:  Hum Mol Genet       Date:  2018-09-15       Impact factor: 6.150

Review 9.  Genetics of lymphatic anomalies.

Authors:  Pascal Brouillard; Laurence Boon; Miikka Vikkula
Journal:  J Clin Invest       Date:  2014-03-03       Impact factor: 14.808

Review 10.  Lymphatic vessel abnormalities arising from disorders of Ras signal transduction.

Authors:  Eva M Sevick-Muraca; Philip D King
Journal:  Trends Cardiovasc Med       Date:  2013-11-01       Impact factor: 6.677

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