Literature DB >> 1802960

Coronal reconstruction images of glucose metabolism in Alzheimer's disease.

H Fukuyama1, K Harada, H Yamauchi, T Miyoshi, S Yamaguchi, J Kimura, M Kameyama, M Senda, Y Yonekura, J Konishi.   

Abstract

Coronal images of position emission tomography (PET) by 18F-labelled deoxyglucose reconstructed from interpolated scan in Alzheimer's disease clearly indicated suppression of glucose metabolism in the parietal lobe and lateral part of the temporal lobes, compared to normal controls. The medial part of the temporal lobes in Alzheimer's disease did show a mildly lower glucose metabolic rate than that in normal controls; therefore it is suggested that the parietal and lateral parts of the temporal lobes are primary affected regions in Alzheimer's disease, not the medial part of the temporal lobe, including the hippocampus.

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Year:  1991        PMID: 1802960     DOI: 10.1016/0022-510x(91)90249-7

Source DB:  PubMed          Journal:  J Neurol Sci        ISSN: 0022-510X            Impact factor:   3.181


  3 in total

Review 1.  Is ammonia a pathogenetic factor in Alzheimer's disease?

Authors:  N Seiler
Journal:  Neurochem Res       Date:  1993-03       Impact factor: 3.996

2.  Cognitive impairment in Alzheimer's disease correlates with ventricular width and atrophy-corrected cortical glucose metabolism.

Authors:  I Slansky; K Herholz; U Pietrzyk; J Kessler; M Grond; R Mielke; W D Heiss
Journal:  Neuroradiology       Date:  1995-05       Impact factor: 2.804

3.  Progression of logopenic variant primary progressive aphasia to apraxia and semantic memory deficits.

Authors:  Michitaka Funayama; Yoshitaka Nakagawa; Yoko Yamaya; Fumihiro Yoshino; Masaru Mimura; Motoichiro Kato
Journal:  BMC Neurol       Date:  2013-11-01       Impact factor: 2.474

  3 in total

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