Literature DB >> 18005737

Orchestration of dysregulated epithelial turnover by a manipulative pathogen.

Lydia E Wroblewski1, Richard M Peek.   

Abstract

Chronic Helicobacter pylori infection is the strongest known risk factor for the development of gastric cancer. In order to survive and propagate under the harsh conditions of the gastric niche, this pathogen has evolved numerous means to interact with host epithelium. In this issue, Mimuro et al. shed light on mechanisms by which Helicobacter pylori attenuates apoptosis in the gastric epithelium to facilitate its persistence within the human stomach.

Entities:  

Mesh：

Year:  2007        PMID： 18005737     DOI： 10.1016/j.chom.2007.09.011

Source DB:  PubMed          Journal:  Cell Host Microbe        ISSN： 1931-3128            Impact factor:   21.023


  4 in total

1.  High-resolution computational modeling of immune responses in the gut.

Authors:  Meghna Verma; Josep Bassaganya-Riera; Andrew Leber; Nuria Tubau-Juni; Stefan Hoops; Vida Abedi; Xi Chen; Raquel Hontecillas
Journal:  Gigascience       Date:  2019-06-01       Impact factor: 6.524

Review 2.  Helicobacter pylori and gastric cancer: factors that modulate disease risk.

Authors:  Lydia E Wroblewski; Richard M Peek; Keith T Wilson
Journal:  Clin Microbiol Rev       Date:  2010-10       Impact factor: 26.132

3.  Acetylation of apurinic/apyrimidinic endonuclease-1 regulates Helicobacter pylori-mediated gastric epithelial cell apoptosis.

Authors:  Asima Bhattacharyya; Ranajoy Chattopadhyay; Brent R Burnette; Janet V Cross; Sankar Mitra; Peter B Ernst; Kishor K Bhakat; Sheila E Crowe
Journal:  Gastroenterology       Date:  2009-06       Impact factor: 22.682

4.  Identification of Desmoglein-2 as a novel target of Helicobacter pylori HtrA in epithelial cells.

Authors:  Sabine Bernegger; Robert Vidmar; Marko Fonovic; Gernot Posselt; Boris Turk; Silja Wessler
Journal:  Cell Commun Signal       Date:  2021-11-06       Impact factor: 5.712
  4 in total

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