Literature DB >> 1796771

[Rebound nystagmus in normal subjects].

T Suzuki1.   

Abstract

Rebound nystagmus represents a disorder of ocular movement in patients with cerebellar lesions. It was reported, however, that it occurred in normal subjects in darkness. Jerk nystagmus, amplitude of 0.5-2 deg and frequency of less than 2Hz, was evoked after prolonged eccentric fixation and fatigue within about 5 to 15 seconds in 7 normal subjects of the experiment. The nystagmus increased depending on the eccentricity and duration of gaze holding. It also depended on the orbital position 10deg left, 0 deg, 10 deg right after eccentric gaze. It is suggested that rebound nystagmus resulted from the leaky neural integrator of the brainstem, and a shift of the null position in the direction of the eccentric gaze. These two components may cause different types of nystagmus by various combinations.

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Year:  1991        PMID: 1796771

Source DB:  PubMed          Journal:  Nippon Ganka Gakkai Zasshi        ISSN: 0029-0203


  2 in total

1.  "Dumping" of rebound nystagmus and optokinetic afternystagmus in humans.

Authors:  S T Chung; H E Bedell
Journal:  Exp Brain Res       Date:  1995       Impact factor: 1.972

2.  PRMT6-mediated H3R2me2a guides Aurora B to chromosome arms for proper chromosome segregation.

Authors:  Seul Kim; Nam Hyun Kim; Ji Eun Park; Jee Won Hwang; Nayeon Myung; Ki-Tae Hwang; Young A Kim; Chang-Young Jang; Yong Kee Kim
Journal:  Nat Commun       Date:  2020-01-30       Impact factor: 14.919

  2 in total

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