Literature DB >> 17954171

Ischemic postconditioning prevents ischemic acute renal failure.

I Szwarc1, S Soullier, N Gayrard, C Mejean, G Mourad, A Argiles.   

Abstract

INTRODUCTION: Delayed graft function (DGF), a frequent complication after kidney transplantation, decreases graft survival. Ischemia/reperfusion (I/R) injuries play a major role in DGF pathophysiology. Because ischemic postconditioning (IP) is efficient to prevent myocardial I/R injuries and reduce infarct size, we sought to describe renal effects of IP.
MATERIALS AND METHODS: Swiss mice were divided into three groups after left nephrectomy. Thirty minutes of right kidney ischemia followed by three cycles of 30 seconds of ischemia and reperfusion (IP group: n = 12) versus immediate reperfusion (n = 7). Left nephrectomized and right kidney sham operated mice were used as control groups (n = 6). Mice were followed for an 8-day survival analysis. Serum levels of creatinine and protein as well as weights were determined 2 days before and at days 2 and 8 after surgery.
RESULTS: IP improved kidney function on day 2; the mean serum creatinine level was 1.25 +/- 0.71 versus 2.9 +/- 1.3 mg/dL in the immediate reperfusion group (P < .02). We also observed a trend toward increased animal survival (25% vs. 0% in the immediate reperfusion group; P = .10). Despite a significant increase in proteinuria among all groups, there was no significant difference.
CONCLUSION: In a mouse model, IP seems to prevent postischemic acute renal failure after 30 minutes of kidney ischemia.

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Year:  2007        PMID: 17954171     DOI: 10.1016/j.transproceed.2007.08.026

Source DB:  PubMed          Journal:  Transplant Proc        ISSN: 0041-1345            Impact factor:   1.066


  11 in total

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