Literature DB >> 17920279

The PI3K inhibitor arsenal: choose your weapon!

Tom Crabbe1, Melanie J Welham, Stephen G Ward.   

Abstract

Owing to its widespread activation in inflammation and cancer, a growing appreciation of the therapeutic potential of inhibitors of the phosphoinositide 3-kinase (PI3K) pathway has stimulated intense interest in compounds with suitable pharmacological profiles. These are primarily directed toward PI3K itself. However, as class I PI3Ks are also essential for a range of normal physiological processes, broad spectrum PI3K inhibition could be poorly tolerated. In recent years, patents describing a new generation of PI3K inhibitors have started to appear, with a particular focus on the development of compounds with enhanced isoform selectivity for use as anti-cancer and anti-inflammatory therapies. However, challenges remain for the efforts to pharmacologically target this enzyme family in a successful manner.

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Year:  2007        PMID: 17920279     DOI: 10.1016/j.tibs.2007.09.001

Source DB:  PubMed          Journal:  Trends Biochem Sci        ISSN: 0968-0004            Impact factor:   13.807


  28 in total

Review 1.  Drugging the PI3 kinome: from chemical tools to drugs in the clinic.

Authors:  Paul Workman; Paul A Clarke; Florence I Raynaud; Rob L M van Montfort
Journal:  Cancer Res       Date:  2010-02-23       Impact factor: 12.701

2.  p110gamma and p110delta isoforms of phosphoinositide 3-kinase differentially regulate natural killer cell migration in health and disease.

Authors:  Aurore Saudemont; Fabien Garçon; Hakim Yadi; Marta Roche-Molina; Nayoung Kim; Anne Segonds-Pichon; Alfonso Martín-Fontecha; Klaus Okkenhaug; Francesco Colucci
Journal:  Proc Natl Acad Sci U S A       Date:  2009-03-18       Impact factor: 11.205

3.  PI(3) kinases: revealing the delta lady.

Authors:  Paul Workman; Rob L M van Montfort
Journal:  Nat Chem Biol       Date:  2010-02       Impact factor: 15.040

4.  Human neuronal cells possess functional cytoplasmic and TLR-mediated innate immune pathways influenced by phosphatidylinositol-3 kinase signaling.

Authors:  Daniel C Peltier; Allison Simms; Jocelyn R Farmer; David J Miller
Journal:  J Immunol       Date:  2010-05-14       Impact factor: 5.422

5.  Irradiation with heavy-ion particles changes the cellular distribution of human histone acetyltransferase HAT1.

Authors:  Emily A Lebel; Petra Boukamp; Stefan T Tafrov
Journal:  Mol Cell Biochem       Date:  2010-02-11       Impact factor: 3.396

Review 6.  Programmed cell death pathways in cancer: a review of apoptosis, autophagy and programmed necrosis.

Authors:  L Ouyang; Z Shi; S Zhao; F-T Wang; T-T Zhou; B Liu; J-K Bao
Journal:  Cell Prolif       Date:  2012-10-03       Impact factor: 6.831

7.  Relative quantification of PIK3CA gene expression level in fine-needle aspiration biopsy thyroid specimens collected from patients with papillary thyroid carcinoma and non-toxic goitre by real-time RT-PCR.

Authors:  Katarzyna Wojciechowska-Durczyńska; Kinga Krawczyk-Rusiecka; Anna Cyniak-Magierska; Arkadiusz Zygmunt; Elzbieta Gałecka; Andrzej Lewiński
Journal:  Thyroid Res       Date:  2010-08-30

8.  Natural sphingadienes inhibit Akt-dependent signaling and prevent intestinal tumorigenesis.

Authors:  Henrik Fyrst; Babak Oskouian; Padmavathi Bandhuvula; Yaqiong Gong; Hoe Sup Byun; Robert Bittman; Andrew R Lee; Julie D Saba
Journal:  Cancer Res       Date:  2009-12-15       Impact factor: 12.701

9.  Activation of phosphatidylinositol 3-kinase signaling promotes aberrant pituitary growth in a mouse model of thyroid-stimulating hormone-secreting pituitary tumors.

Authors:  Changxue Lu; Mark C Willingham; Fumihiko Furuya; Sheue-Yann Cheng
Journal:  Endocrinology       Date:  2008-03-20       Impact factor: 4.736

Review 10.  Deconstructing feedback-signaling networks to improve anticancer therapy with mTORC1 inhibitors.

Authors:  Arkaitz Carracedo; Jose Baselga; Pier Paolo Pandolfi
Journal:  Cell Cycle       Date:  2008-12-22       Impact factor: 4.534

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