Literature DB >> 17851929

Clinical characteristics and genotype-phenotype correlation of hearing loss patients with SLC26A4 mutations.

Hiroaki Suzuki1, Aki Oshima, Koji Tsukamoto, Satoko Abe, Kozo Kumakawa, Kyoko Nagai, Hitoshi Satoh, Yukihiko Kanda, Satoshi Iwasaki, Shin-ichi Usami.   

Abstract

CONCLUSIONS: The present study confirmed the clinical characteristics of patients with SLC26A4 mutations: congenital, fluctuating, and progressive hearing loss usually associated with vertigo and/or goiter during long-term follow-up. This clarification should help to facilitate appropriate genetic counseling and proper medical management for patients with these mutations, but there was no particular genotype-phenotype correlation among them, suggesting that other factors may contribute to such variability.
OBJECTIVES: Due to the wide range of phenotypes caused by SLC26A4 mutations, there is controversy with regard to genotype-phenotype correlation. The present study was performed: (1) to determine phenotypic range in patients with biallelic SLC26A4 mutations, and (2) to evaluate whether possible genotype-phenotype correlation exists. SUBJECTS AND METHODS: Phenotypes in 39 hearing loss patients with SLC26A4 mutations were summarized and genotype-phenotype correlation was analyzed.
RESULTS: Hearing level varied in the individuals from mild to profound severity. Most of the patients had fluctuating and progressive hearing loss that may have been of prelingual onset. Twenty-four (70.6%) patients had episodes of vertigo, and 10 (27.8%) patients had goiter, which had appeared at age 12 or older. In contrast to such phenotypic variabilities, no apparent correlation was found between these phenotypes and their genotypes.

Entities:  

Mesh:

Substances:

Year:  2007        PMID: 17851929     DOI: 10.1080/00016480701258739

Source DB:  PubMed          Journal:  Acta Otolaryngol        ISSN: 0001-6489            Impact factor:   1.494


  16 in total

Review 1.  Integration of human and mouse genetics reveals pendrin function in hearing and deafness.

Authors:  Amiel A Dror; Zippora Brownstein; Karen B Avraham
Journal:  Cell Physiol Biochem       Date:  2011-11-18

Review 2.  The role of pendrin in the development of the murine inner ear.

Authors:  Philine Wangemann
Journal:  Cell Physiol Biochem       Date:  2011-11-18

3.  SLC26A4 mutation testing for hearing loss associated with enlargement of the vestibular aqueduct.

Authors:  Taku Ito; Julie Muskett; Parna Chattaraj; Byung Yoon Choi; Kyu Yup Lee; Christopher K Zalewski; Kelly A King; Xiangming Li; Philine Wangemann; Thomas Shawker; Carmen C Brewer; Seth L Alper; Andrew J Griffith
Journal:  World J Otorhinolaryngol       Date:  2013-05-28

4.  Exploring the missing heritability in subjects with hearing loss, enlarged vestibular aqueducts, and a single or no pathogenic SLC26A4 variant.

Authors:  Jeroen J Smits; Suzanne E de Bruijn; Cornelis P Lanting; Jaap Oostrik; Luke O'Gorman; Tuomo Mantere; Frans P M Cremers; Susanne Roosing; Helger G Yntema; Erik de Vrieze; Ronny Derks; Alexander Hoischen; Sjoert A H Pegge; Kornelia Neveling; Ronald J E Pennings; Hannie Kremer
Journal:  Hum Genet       Date:  2021-08-19       Impact factor: 5.881

5.  Efficient molecular genetic diagnosis of enlarged vestibular aqueducts in East Asians.

Authors:  Byung Yoon Choi; Andrew K Stewart; Katherine K Nishimura; Won Jae Cha; Moon-Woo Seong; Sung Sup Park; Seung Won Kim; Yang Sook Chun; Jong Woo Chung; Shi-Nae Park; Sun O Chang; Chong-Sun Kim; Seth L Alper; Andrew J Griffith; Seung-Ha Oh
Journal:  Genet Test Mol Biomarkers       Date:  2009-10

6.  Different cortical metabolic activation by visual stimuli possibly due to different time courses of hearing loss in patients with GJB2 and SLC26A4 mutations.

Authors:  Hideaki Moteki; Yasushi Naito; Keizo Fujiwara; Ryosuke Kitoh; Shin-ya Nishio; Kazuhiro Oguchi; Yutaka Takumi; Shin-ichi Usami
Journal:  Acta Otolaryngol       Date:  2011-07-05       Impact factor: 1.494

7.  Failure of fluid absorption in the endolymphatic sac initiates cochlear enlargement that leads to deafness in mice lacking pendrin expression.

Authors:  Hyoung-Mi Kim; Philine Wangemann
Journal:  PLoS One       Date:  2010-11-17       Impact factor: 3.240

8.  Establishment of a knock-in mouse model with the SLC26A4 c.919-2A>G mutation and characterization of its pathology.

Authors:  Ying-Chang Lu; Chen-Chi Wu; Wen-Sheng Shen; Ting-Hua Yang; Te-Huei Yeh; Pei-Jer Chen; I-Shing Yu; Shu-Wha Lin; Jau-Min Wong; Qing Chang; Xi Lin; Chuan-Jen Hsu
Journal:  PLoS One       Date:  2011-07-21       Impact factor: 3.240

9.  Simultaneous screening of multiple mutations by invader assay improves molecular diagnosis of hereditary hearing loss: a multicenter study.

Authors:  Shin-ichi Usami; Shin-ya Nishio; Makoto Nagano; Satoko Abe; Toshikazu Yamaguchi
Journal:  PLoS One       Date:  2012-02-24       Impact factor: 3.240

10.  Mutation spectrum and genotype-phenotype correlation of hearing loss patients caused by SLC26A4 mutations in the Japanese: a large cohort study.

Authors:  Maiko Miyagawa; Shin-Ya Nishio; Shin-Ichi Usami
Journal:  J Hum Genet       Date:  2014-03-06       Impact factor: 3.172

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.