Literature DB >> 17805550

Caspase-3 activity, response to chemotherapy and clinical outcome in patients with colon cancer.

Javier de Oca1, Daniel Azuara, Raquel Sanchez-Santos, Matilde Navarro, Gabriel Capella, Victor Moreno, Anna Sola, Georgina Hotter, Sebastiano Biondo, Alfonso Osorio, Joan Martí-Ragué, Antoni Rafecas.   

Abstract

BACKGROUND AND AIMS: The prognostic value of the degree of apoptosis in colorectal cancer is controversial. This study evaluates the putative clinical usefulness of measuring caspase-3 activity as a prognostic factor in colonic cancer patients receiving 5-fluoracil adjuvant chemotherapy.
MATERIALS AND METHODS: We evaluated caspase-3-like protease activity in tumours and in normal colon tissue. Specimens were studied from 54 patients. These patients had either stage III cancer (Dukes stage C) or high-risk stage II cancer (Dukes stage B2 with invasion of adjacent organs, lymphatic or vascular infiltration or carcinoembryonic antigen [CEA] >5). Median follow-up was 73 months. Univariate analysis was performed previously to explore the relation of different variables (age, sex, preoperative CEA, tumour size, Dukes stage, vascular invasion, lymphatic invasion, caspase-3 activity in tumour and caspase-3 activity in normal mucosa) as prognostic factors of tumour recurrence after chemotherapy treatment. Subsequently, a multivariate Cox regression model was performed.
RESULTS: Median values of caspase-3 activity in tumours were more than twice those in normal mucosa (88.1 vs 40.6 U, p=0.001), showing a statistically significant correlation (r=0.34). Significant prognostic factors of recurrence in multivariate analysis were: male sex (odds ratio, OR=3.53 [1.13-10.90], p=0.02), age (OR=1.09 [1.01-1.18], p=0.03), Dukes stage (OR=1.93 [1.01-3.70]), caspase-3 activity in normal mucosa (OR=1.02 [1.01-1.04], p=0.017) and caspase-3 activity in tumour (OR=1.02 [1.01-1.03], p=0.013).
CONCLUSION: Low caspase-3 activity in the normal mucosa and tumour are independent prognostic factors of tumour recurrence in patients receiving adjuvant 5-fluoracil-based treatment in colon cancer, correlating with poor disease-free survival and higher recurrence rate.

Entities:  

Mesh:

Substances:

Year:  2007        PMID: 17805550     DOI: 10.1007/s00384-007-0362-3

Source DB:  PubMed          Journal:  Int J Colorectal Dis        ISSN: 0179-1958            Impact factor:   2.571


  21 in total

Review 1.  Apoptosis. Biochemical events and relevance to cancer chemotherapy.

Authors:  S Sen; M D'Incalci
Journal:  FEBS Lett       Date:  1992-07-27       Impact factor: 4.124

Review 2.  Changes in apoptosis during the development of colorectal cancer: a systematic review of the literature.

Authors:  J J Koornstra; S de Jong; H Hollema; E G E de Vries; J H Kleibeuker
Journal:  Crit Rev Oncol Hematol       Date:  2003-01       Impact factor: 6.312

3.  Apoptosis and immunohistochemical bcl-2 expression in colorectal adenomas and carcinomas. Aspects of carcinogenesis and prognostic significance.

Authors:  G B Baretton; J Diebold; G Christoforis; M Vogt; C Müller; K Dopfer; K Schneiderbanger; M Schmidt; U Löhrs
Journal:  Cancer       Date:  1996-01-15       Impact factor: 6.860

4.  Pathological and genetic correlates of apoptosis in the progression of colorectal neoplasia.

Authors:  N Hawkins; J Lees; R Hargrave; T O'Connor; A Meagher; R Ward
Journal:  Tumour Biol       Date:  1997

5.  The activity of caspase-3-like proteases is elevated during the development of colorectal carcinoma.

Authors:  L Leonardos; L M Butler; P J Hewett; P D Zalewski; P A Cowled
Journal:  Cancer Lett       Date:  1999-08-23       Impact factor: 8.679

6.  Apoptosis as a cellular predictor for histopathologic response to neoadjuvant radiochemotherapy in patients with rectal cancer.

Authors:  Claus Rödel; Gerhard G Grabenbauer; Thomas Papadopoulos; Marc Bigalke; Klaus Günther; Christoph Schick; Andrea Peters; Rolf Sauer; Franz Rödel
Journal:  Int J Radiat Oncol Biol Phys       Date:  2002-02-01       Impact factor: 7.038

7.  Inhibition of apoptosis by survivin predicts shorter survival rates in colorectal cancer.

Authors:  H Kawasaki; D C Altieri; C D Lu; M Toyoda; T Tenjo; N Tanigawa
Journal:  Cancer Res       Date:  1998-11-15       Impact factor: 12.701

8.  Imbalance between proliferation and apoptosis in the development of colorectal carcinoma.

Authors:  X Hao; M Du; A E Bishop; I C Talbot
Journal:  Virchows Arch       Date:  1998-12       Impact factor: 4.064

Review 9.  Apoptosis. Its significance in cancer and cancer therapy.

Authors:  J F Kerr; C M Winterford; B V Harmon
Journal:  Cancer       Date:  1994-04-15       Impact factor: 6.860

10.  Inhibition of apoptosis during development of colorectal cancer.

Authors:  A Bedi; P J Pasricha; A J Akhtar; J P Barber; G C Bedi; F M Giardiello; B A Zehnbauer; S R Hamilton; R J Jones
Journal:  Cancer Res       Date:  1995-05-01       Impact factor: 12.701

View more
  9 in total

Review 1.  Prognostic value of Caspase-3 expression in cancers of digestive tract: a meta-analysis and systematic review.

Authors:  Hao Chen; Xia Yang; Zhenbo Feng; Ruixue Tang; Fanghui Ren; Kanglai Wei; Gang Chen
Journal:  Int J Clin Exp Med       Date:  2015-07-15

2.  Bortezomib inhibits cell proliferation in prostate cancer.

Authors:  Ren-Ping Zheng; Wei Wang; Chuan-Dong Wei
Journal:  Exp Ther Med       Date:  2015-07-03       Impact factor: 2.447

3.  Preoperative radiochemotherapy is successful also in patients with locally advanced rectal cancer who have intrinsically high apoptotic tumours.

Authors:  M J E M Gosens; R C Dresen; H J T Rutten; G A P Nieuwenhuijzen; J A W M van der Laak; H Martijn; I Tan-Go; I D Nagtegaal; A J C van den Brule; J H J M van Krieken
Journal:  Ann Oncol       Date:  2008-07-29       Impact factor: 32.976

4.  Targeting the XIAP/caspase-7 complex selectively kills caspase-3-deficient malignancies.

Authors:  Yuan-Feng Lin; Tsung-Ching Lai; Chih-Kang Chang; Chi-Long Chen; Ming-Shyan Huang; Chih-Jen Yang; Hon-Ge Liu; Jhih-Jhong Dong; Yi-An Chou; Kuo-Hsun Teng; Shih-Hsun Chen; Wei-Ting Tian; Yi-Hua Jan; Michael Hsiao; Po-Huang Liang
Journal:  J Clin Invest       Date:  2013-08-27       Impact factor: 14.808

5.  Targeting procaspase-3 with WF-208, a novel PAC-1 derivative, causes selective cancer cell apoptosis.

Authors:  Fangyang Wang; Yajing Liu; Lihui Wang; Jingyu Yang; Yanfang Zhao; Nannan Wang; Qi Cao; Ping Gong; Chunfu Wu
Journal:  J Cell Mol Med       Date:  2015-03-08       Impact factor: 5.310

6.  Calotropin regulates the apoptosis of non‑small cell cancer by regulating the cytotoxic T‑lymphocyte associated antigen 4‑mediated TGF‑β/ERK signaling pathway.

Authors:  Lu Tian; Xiao-Hong Xie; Ze-Hao Zhu
Journal:  Mol Med Rep       Date:  2018-04-05       Impact factor: 2.952

7.  Expression Level of Caspase Genes in Colorectal Cancer

Authors:  Milad Asadi; Dariush Shanehbandi; Touraj Asvadi Kermani; Zohreh Sanaat; Venus Zafari; Shahriyar Hashemzadeh
Journal:  Asian Pac J Cancer Prev       Date:  2018-05-26

8.  Clinical significance of stromal apoptosis in colorectal cancer.

Authors:  P J Koelink; C F M Sier; D W Hommes; C B H W Lamers; H W Verspaget
Journal:  Br J Cancer       Date:  2009-08-04       Impact factor: 7.640

9.  MicroRNA-224 is implicated in lung cancer pathogenesis through targeting caspase-3 and caspase-7.

Authors:  Ri Cui; Taewan Kim; Matteo Fassan; Wei Meng; Hui-Lung Sun; Young-Jun Jeon; Caterina Vicentini; Esmerina Tili; Yong Peng; Aldo Scarpa; Guang Liang; Yong Kui Zhang; Arnab Chakravarti; Carlo M Croce
Journal:  Oncotarget       Date:  2015-09-08
  9 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.