Literature DB >> 17674321

Intralesional expression of mRNA of interferon- gamma , tumor necrosis factor- alpha , interleukin-10, nitric oxide synthase, indoleamine-2,3-dioxygenase, and RANTES is a major immune effector in Mediterranean spotted fever rickettsiosis.

Rita de Sousa1, Nahed Ismail, Sónia Dória Nobrega, Ana França, Mário Amaro, Margarida Anes, José Poças, Ricardo Coelho, Jorge Torgal, Fátima Bacellar, David H Walker.   

Abstract

BACKGROUND: The mechanisms of immunity to Rickettsia conorii that have been elucidated in mouse models have not been evaluated in human tissues.
METHODS: In this study, quantitative real-time polymerase chain reaction was used to determine the levels of expression of inflammatory and immune mediators in skin-biopsy samples collected from 23 untreated patients with Mediterranean spotted fever (MSF).
RESULTS: In all 23 patients, the levels of intralesional expression of mRNA of tumor necrosis factor (TNF)- alpha , interferon (IFN)- gamma , interleukin (IL)-10, RANTES, and indoleamine-2,3-dioxygenase (IDO), an enzyme involved in limiting rickettsial growth by tryptophan degradation, were higher than those in control subjects; 6 of the 23 patients had high levels of inducible nitric oxide synthase (iNOS), a source of microbicidal nitric oxide. Positive correlations between TNF- alpha , IFN- gamma , iNOS, IDO, and mild/moderate MSF suggest that type 1 polarization plays a protective role. Significantly higher levels of intralesional expression of IL-10 mRNA were inversely correlated with levels of intralesional expression of IFN- gamma mRNA and TNF- alpha mRNA. The mRNA-expression level of the chemokine RANTES was significantly higher in patients with severe MSF.
CONCLUSION: Mild/moderate MSF is associated with a strong and balanced intralesional proinflammatory and anti-inflammatory response, with a dominant type 1 immunity, whereas severe MSF is associated with increased expression of chemokine mRNA. Whether these factors are simply correlates of mild and severe MSF or contribute to antirickettsial immunity and pathogenesis remains to be determined.

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Year:  2007        PMID: 17674321     DOI: 10.1086/519739

Source DB:  PubMed          Journal:  J Infect Dis        ISSN: 0022-1899            Impact factor:   5.226


  21 in total

Review 1.  Host, pathogen and treatment-related prognostic factors in rickettsioses.

Authors:  E Botelho-Nevers; D Raoult
Journal:  Eur J Clin Microbiol Infect Dis       Date:  2011-04-26       Impact factor: 3.267

2.  CD4+ CD25+ Foxp3- T-regulatory cells produce both gamma interferon and interleukin-10 during acute severe murine spotted fever rickettsiosis.

Authors:  Rong Fang; Nahed Ismail; Thomas Shelite; David H Walker
Journal:  Infect Immun       Date:  2009-06-29       Impact factor: 3.441

Review 3.  Recent molecular insights into rickettsial pathogenesis and immunity.

Authors:  Sanjeev K Sahni; Hema P Narra; Abha Sahni; David H Walker
Journal:  Future Microbiol       Date:  2013-10       Impact factor: 3.165

4.  Susceptibility of inbred mice to Rickettsia parkeri.

Authors:  Britton J Grasperge; Kathryn E Reif; Timothy D Morgan; Piyanate Sunyakumthorn; Joseph Bynog; Christopher D Paddock; Kevin R Macaluso
Journal:  Infect Immun       Date:  2012-03-05       Impact factor: 3.441

5.  Natural killer cells promote tissue injury and systemic inflammatory responses during fatal Ehrlichia-induced toxic shock-like syndrome.

Authors:  Heather L Stevenson; Mark D Estes; Nagaraja R Thirumalapura; David H Walker; Nahed Ismail
Journal:  Am J Pathol       Date:  2010-07-08       Impact factor: 4.307

Review 6.  Pathogenesis of Rickettsial Diseases: Pathogenic and Immune Mechanisms of an Endotheliotropic Infection.

Authors:  Abha Sahni; Rong Fang; Sanjeev K Sahni; David H Walker
Journal:  Annu Rev Pathol       Date:  2018-08-27       Impact factor: 23.472

Review 7.  The realities of biodefense vaccines against Rickettsia.

Authors:  David H Walker
Journal:  Vaccine       Date:  2009-11-05       Impact factor: 3.641

8.  Host- and microbe-related risk factors for and pathophysiology of fatal Rickettsia conorii infection in Portuguese patients.

Authors:  Rita de Sousa; Ana França; Sónia Dória Nòbrega; Adelaide Belo; Mario Amaro; Tiago Abreu; José Poças; Paula Proença; José Vaz; Jorge Torgal; Fátima Bacellar; Nahed Ismail; David H Walker
Journal:  J Infect Dis       Date:  2008-08-15       Impact factor: 5.226

9.  Relevance of gamma interferon, tumor necrosis factor alpha, and interleukin-10 gene polymorphisms to susceptibility to Mediterranean spotted fever.

Authors:  Giusi Irma Forte; Letizia Scola; Gabriella Misiano; Salvatore Milano; Pasquale Mansueto; Giustina Vitale; Fiamma Bellanca; Maria Sanacore; Loredana Vaccarino; Giovan Battista Rini; Calogero Caruso; Enrico Cillari; Domenico Lio; Serafino Mansueto
Journal:  Clin Vaccine Immunol       Date:  2009-04-22

Review 10.  Host-cell interactions with pathogenic Rickettsia species.

Authors:  Sanjeev K Sahni; Elena Rydkina
Journal:  Future Microbiol       Date:  2009-04       Impact factor: 3.165

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