Literature DB >> 17627584

Tumor suppression by DNA base excision repair.

G Frosina1.   

Abstract

Base excision repair (BER) is the main pathway for repair of endogenous DNA damage. It was expected that different tumor types could derive from BER defects but to date this link is elusive. In vitro and molecular epidemiology studies may be used to unravel this issue.

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Year:  2007        PMID： 17627584     DOI： 10.2174/138955707781024544

Source DB:  PubMed          Journal:  Mini Rev Med Chem        ISSN： 1389-5575            Impact factor:   3.862


  3 in total

1.  XRCC1 and base excision repair balance in response to nitric oxide.

Authors:  James T Mutamba; David Svilar; Somsak Prasongtanakij; Xiao-Hong Wang; Ying-Chih Lin; Peter C Dedon; Robert W Sobol; Bevin P Engelward
Journal:  DNA Repair (Amst)       Date:  2011-10-29

2.  Fat10 is an epigenetic marker for liver preneoplasia in a drug-primed mouse model of tumorigenesis.

Authors:  Joan Oliva; Fawzia Bardag-Gorce; Barbara A French; Jun Li; Laron McPhaul; Fataneh Amidi; Jeniffer Dedes; Amir Habibi; Sheila Nguyen; Samuel W French
Journal:  Exp Mol Pathol       Date:  2008-01-11       Impact factor: 3.362

3.  Deoxyinosine repair in nuclear extracts of human cells.

Authors:  Chia-Chia Lee; Ya-Chien Yang; Steven D Goodman; Shi Chen; Teng-Yung Huang; Wern-Cherng Cheng; Liang-In Lin; Woei-Horng Fang
Journal:  Cell Biosci       Date:  2015-09-08       Impact factor: 7.133
  3 in total

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