Literature DB >> 17618940

Dysbindin (DTNBP1) and the biogenesis of lysosome-related organelles complex 1 (BLOC-1): main and epistatic gene effects are potential contributors to schizophrenia susceptibility.

Derek W Morris1, Kevin Murphy, Niamh Kenny, Shaun M Purcell, Kevin A McGhee, Siobhan Schwaiger, Jeanne-Marie Nangle, Gary Donohoe, Sarah Clarke, Paul Scully, John Quinn, David Meagher, Patrizia Baldwin, Niall Crumlish, Eadbhard O'Callaghan, John L Waddington, Michael Gill, Aiden P Corvin.   

Abstract

BACKGROUND: The DTNBP1 gene, encoding dysbindin, has been strongly implicated in schizophrenia (SZ) susceptibility by a series of independent genetic association and gene expression studies. Among its known functions, dysbindin is part of a protein complex, termed the biogenesis of lysosome-related organelles complex 1 (BLOC-1), the molecular components of which might be involved in the regulation of vesicular trafficking and dendrite branching.
METHODS: A systematic investigation of the other seven BLOC-1 genes (MUTED, PLDN, CNO, SNAPAP, BLOC1S1, BLOC1S2, and BLOC1S3) for evidence of association with SZ was undertaken in a sample of 373 SZ cases and 812 control subjects. Possible epistasis between combinations of BLOC-1 genes, including DTNBP1, was tested with a novel method of investigating for gene-gene interaction. Quality control measures were incorporated into genotyping strategy, and all results were corrected for multiple testing to prevent false positive results.
RESULTS: We identified significant evidence of association between BLOC1S3 and SZ (odds ratio = 1.45, confidence interval = 1.13-1.86, p = .0028, corrected p = .0389). We also report evidence for epistatic interaction between DTNBP1 and MUTED contributing to SZ in the absence of a significant main effect at MUTED (p = .0009, corrected p = .0252). Single marker and epistasis results remained significant after correction for multiple testing.
CONCLUSIONS: Together these data provide evidence for the involvement of the BLOC-1 protein complex in SZ pathogenesis.

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Year:  2007        PMID: 17618940     DOI: 10.1016/j.biopsych.2006.12.025

Source DB:  PubMed          Journal:  Biol Psychiatry        ISSN: 0006-3223            Impact factor:   13.382


  21 in total

Review 1.  Cell biology of the BLOC-1 complex subunit dysbindin, a schizophrenia susceptibility gene.

Authors:  Ariana P Mullin; Avanti Gokhale; Jennifer Larimore; Victor Faundez
Journal:  Mol Neurobiol       Date:  2011-04-26       Impact factor: 5.590

2.  Dysbindin-1C is required for the survival of hilar mossy cells and the maturation of adult newborn neurons in dentate gyrus.

Authors:  Hao Wang; Yefeng Yuan; Zhao Zhang; Hui Yan; Yaqin Feng; Wei Li
Journal:  J Biol Chem       Date:  2014-08-25       Impact factor: 5.157

3.  Disrupted-in-schizophrenia 1 (DISC1) regulates dysbindin function by enhancing its stability.

Authors:  Seol-Ae Lee; Seong-Mo Kim; Bo Kyoung Suh; Hwa-Young Sun; Young-Un Park; Ji-Ho Hong; Cana Park; Minh Dang Nguyen; Koh-Ichi Nagata; Joo-Yeon Yoo; Sang Ki Park
Journal:  J Biol Chem       Date:  2015-01-29       Impact factor: 5.157

Review 4.  The road to lysosome-related organelles: Insights from Hermansky-Pudlak syndrome and other rare diseases.

Authors:  Shanna L Bowman; Jing Bi-Karchin; Linh Le; Michael S Marks
Journal:  Traffic       Date:  2019-06       Impact factor: 6.215

5.  A human-specific AS3MT isoform and BORCS7 are molecular risk factors in the 10q24.32 schizophrenia-associated locus.

Authors:  Ming Li; Andrew E Jaffe; Richard E Straub; Ran Tao; Joo Heon Shin; Yanhong Wang; Qiang Chen; Chao Li; Yankai Jia; Kazutaka Ohi; Brady J Maher; Nicholas J Brandon; Alan Cross; Joshua G Chenoweth; Daniel J Hoeppner; Huijun Wei; Thomas M Hyde; Ronald McKay; Joel E Kleinman; Daniel R Weinberger
Journal:  Nat Med       Date:  2016-05-09       Impact factor: 53.440

6.  GWA study data mining and independent replication identify cardiomyopathy-associated 5 (CMYA5) as a risk gene for schizophrenia.

Authors:  X Chen; G Lee; B S Maher; A H Fanous; J Chen; Z Zhao; A Guo; E van den Oord; P F Sullivan; J Shi; D F Levinson; P V Gejman; A Sanders; J Duan; M J Owen; N J Craddock; M C O'Donovan; J Blackman; D Lewis; G K Kirov; W Qin; S Schwab; D Wildenauer; K Chowdari; V Nimgaonkar; R E Straub; D R Weinberger; F A O'Neill; D Walsh; M Bronstein; A Darvasi; T Lencz; A K Malhotra; D Rujescu; I Giegling; T Werge; T Hansen; A Ingason; M M Nöethen; M Rietschel; S Cichon; S Djurovic; O A Andreassen; R M Cantor; R Ophoff; A Corvin; D W Morris; M Gill; C N Pato; M T Pato; A Macedo; H M D Gurling; A McQuillin; J Pimm; C Hultman; P Lichtenstein; P Sklar; S M Purcell; E Scolnick; D St Clair; D H R Blackwood; K S Kendler
Journal:  Mol Psychiatry       Date:  2010-09-14       Impact factor: 15.992

7.  Evidence that the BLOC-1 protein dysbindin modulates dopamine D2 receptor internalization and signaling but not D1 internalization.

Authors:  Yukihiko Iizuka; Yoshitatsu Sei; Daniel R Weinberger; Richard E Straub
Journal:  J Neurosci       Date:  2007-11-07       Impact factor: 6.167

8.  Genetic modifiers of abnormal organelle biogenesis in a Drosophila model of BLOC-1 deficiency.

Authors:  Verónica T Cheli; Richard W Daniels; Ruth Godoy; Diego J Hoyle; Vasundhara Kandachar; Marta Starcevic; Julian A Martinez-Agosto; Stephen Poole; Aaron DiAntonio; Vett K Lloyd; Henry C Chang; David E Krantz; Esteban C Dell'Angelica
Journal:  Hum Mol Genet       Date:  2009-12-16       Impact factor: 6.150

9.  Snapin facilitates the synchronization of synaptic vesicle fusion.

Authors:  Ping-Yue Pan; Jin-Hua Tian; Zu-Hang Sheng
Journal:  Neuron       Date:  2009-02-12       Impact factor: 17.173

10.  The dysbindin-containing complex (BLOC-1) in brain: developmental regulation, interaction with SNARE proteins and role in neurite outgrowth.

Authors:  C A Ghiani; M Starcevic; I A Rodriguez-Fernandez; R Nazarian; V T Cheli; L N Chan; J S Malvar; J de Vellis; C Sabatti; E C Dell'Angelica
Journal:  Mol Psychiatry       Date:  2009-06-23       Impact factor: 15.992

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