| Literature DB >> 17582324 |
Abstract
Everyone agrees about how long-term potentiation (LTP) is induced-NMDA receptor activation-but much remains to be learned about how the increase in the strength of a synaptic connection between two neurons is expressed. In this issue of Neuron, Kim et al. report a new form of NMDAR-dependent plasticity that may contribute to LTP: internalization of postsynaptic Kv4.2 potassium channels that mediate transient IA-type outward current in dendrites.Entities:
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Year: 2007 PMID： 17582324 DOI： 10.1016/j.neuron.2007.06.008
Source DB: PubMed Journal: Neuron ISSN： 0896-6273 Impact factor: 17.173