Literature DB >> 17557798

Relation between cognitive dysfunction and reduced vital capacity in amyotrophic lateral sclerosis.

S-M Kim1, K-M Lee, Y-H Hong, K S Park, J-H Yang, H-W Nam, J-J Sung, K-W Lee.   

Abstract

BACKGROUND: Many patients with amyotrophic lateral sclerosis (ALS) with cognitive impairment have fronto-temporal dysfunction. Whereas in some patients with ALS the fronto-temporal dysfunction is undoubtedly due to the degenerative process associated with the disease, in others dysfunction cannot be accounted for by an irreversible degenerative process alone, as it also appears to involve a reversible process. We hypothesised that reduced vital capacity can be a key contributor to the fronto-temporal dysfunction observed in patients with ALS.
OBJECTIVE: To investigate the association between fronto-temporal dysfunction and reduced vital capacity in ALS.
METHODS: 16 consecutive patients who conformed to a diagnosis of definite or probable ALS (El escorial criteria) were grouped by vital capacity, and their clinical characteristics and cognitive functions, including disease duration, attention, executive function and memory, were measured.
RESULTS: Patients with a reduced vital capacity performed significantly poorer in memory retention (p = 0.028), retrieval efficacy (p = 0.003), spoken verbal fluency (p = 0.03) and spoken verbal fluency indexes (p = 0.016) than those with a normal vital capacity.
CONCLUSION: The fronto-temporal dysfunction in ALS might be attributable to potentially reversible secondary effects associated with reduced vital capacity, as well as to the primary degenerative process.

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Year:  2007        PMID: 17557798      PMCID: PMC2095584          DOI: 10.1136/jnnp.2006.111195

Source DB:  PubMed          Journal:  J Neurol Neurosurg Psychiatry        ISSN: 0022-3050            Impact factor:   10.154


  14 in total

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  13 in total

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2.  Patterns of Language Impairment in Early Amyotrophic Lateral Sclerosis.

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3.  Cognitive-behavioral screening reveals prevalent impairment in a large multicenter ALS cohort.

Authors:  Jennifer Murphy; Pam Factor-Litvak; Raymond Goetz; Catherine Lomen-Hoerth; Peter L Nagy; Jonathan Hupf; Jessica Singleton; Susan Woolley; Howard Andrews; Daragh Heitzman; Richard S Bedlack; Jonathan S Katz; Richard J Barohn; Eric J Sorenson; Björn Oskarsson; J Americo M Fernandes Filho; Edward J Kasarskis; Tahseen Mozaffar; Yvonne D Rollins; Sharon P Nations; Andrea J Swenson; Boguslawa A Koczon-Jaremko; Hiroshi Mitsumoto
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5.  The Relationship between Depressive Symptoms, Disease State, and Cognition in Amyotrophic Lateral Sclerosis.

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6.  Capnography for assessing nocturnal hypoventilation and predicting compliance with subsequent noninvasive ventilation in patients with ALS.

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7.  Amyotrophic lateral sclerosis is associated with hypolipidemia at the presymptomatic stage in mice.

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8.  Nocturnal hypoxia in ALS is related to cognitive dysfunction and can occur as clusters of desaturations.

Authors:  Su-Yeon Park; Sung-Min Kim; Jung-Joon Sung; Kyung-Min Lee; Kyung-Seok Park; Sang-Yun Kim; Hyun-woo Nam; Kwang-Woo Lee
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9.  Cognition, behavior, and respiratory function in amyotrophic lateral sclerosis.

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10.  Intermittent hypoxia can aggravate motor neuronal loss and cognitive dysfunction in ALS mice.

Authors:  Sung-Min Kim; Heejaung Kim; Jeong-Seon Lee; Kyung Seok Park; Gye Sun Jeon; Jeeheun Shon; Suk-Won Ahn; Seung Hyun Kim; Kyung Min Lee; Jung-Joon Sung; Kwang-Woo Lee
Journal:  PLoS One       Date:  2013-11-26       Impact factor: 3.240

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