Literature DB >> 17531011

Development of an experimental model of induced bacterial peritonitis in cirrhotic rats with or without ascites.

Elisabet Sánchez1, José Such, Maite Teresa Chiva, Germán Soriano, Teresa Llovet, Javier Mercè, Francisco Sancho, Carlos Muñoz, Xiao-yu Song, Miguel Pérez-Mateo, Joaquín Balanzó, Carlos Guarner.   

Abstract

BACKGROUND: Spontaneous bacterial peritonitis (SBP) is a severe complication of cirrhotic patients associated with a high mortality. AIM: To develop an available experimental model of induced bacterial peritonitis in cirrhosis.
MATERIAL AND METHODS: Sprague-Dawley rats with carbon-tetrachloride-induced cirrhosis with (N=22) or without (N=101) ascites were randomized to receive an intraperitoneal administration of different concentrations of Escherichia coli (E. coli) diluted in 1 mL of sterile water in ascitic rats and in different volumes in nonascitic rats. A subgroup of nonascitic animals received ceftriaxone 4 h after E. coli inoculation. Mortality of rats was evaluated 24 h after bacterial inoculation.
RESULTS: None of the rats receiving sterile water alone and only one infected with 10(7) cfu of E. coli died. Ascitic rats showed a lower mortality rate than nonascitic rats infected with 10(8) or 10(9) cfu of E. coli (P<0.05). Mortality was higher with 10(9) cfu than with 10(8) cfu of E. coli in ascitic (P NS) and nonascitic (P<0.01) rats. A trend was noted to ward higher mortality in nonascitic rats inoculated with 10(8) cfu with increasing water volumes. A marked peritoneal polymorphonuclear cell response was observed 4 h after E. coli injection in both ascitic and nonascitic rats. Antibiotic therapy significantly reduced the mortality rate of rats infected with 10(8) cfu (P<0.01).
CONCLUSIONS: This experimental model of induced bacterial peritonitis in cirrhosis with or without ascites may represent a useful tool for the study of pathogenic events postinfection and for the design of new therapeutic strategies to treat patients with SBP.

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Year:  2007        PMID: 17531011     DOI: 10.1111/j.1572-0241.2007.01182.x

Source DB:  PubMed          Journal:  Am J Gastroenterol        ISSN: 0002-9270            Impact factor:   10.864


  4 in total

1.  Intestinal decontamination inhibits TLR4 dependent fibronectin-mediated cross-talk between stellate cells and endothelial cells in liver fibrosis in mice.

Authors:  Qiang Zhu; Li Zou; Kumaravelu Jagavelu; Douglas A Simonetto; Robert C Huebert; Zhi-Dong Jiang; Herbert L DuPont; Vijay H Shah
Journal:  J Hepatol       Date:  2011-12-13       Impact factor: 25.083

2.  Predictive Value of the Model of End-Stage Liver Disease in Cirrhotic Patients with and without Spontaneous Bacterial Peritonitis.

Authors:  Bledar Kraja; Marsela Sina; Iris Mone; Fatjona Pupuleku; Adriana Babameto; Skerdi Prifti; Genc Burazeri
Journal:  Gastroenterol Res Pract       Date:  2012-02-22       Impact factor: 2.260

3.  Modulation of inflammatory response in a cirrhotic rat model with induced bacterial peritonitis.

Authors:  Elisabet Sánchez; Rubén Francés; Germán Soriano; Beatriz Mirelis; Francesc J Sancho; José Manuel González-Navajas; Carlos Muñoz; Xiao-yu Song; Miguel Pérez-Mateo; José Such; Carlos Guarner
Journal:  PLoS One       Date:  2013-03-20       Impact factor: 3.240

4.  [Development of animal experimental model for bacterial peritonitis].

Authors:  O Montenegro; S Illescas; J C González; D Padilla; P Villarejo; V Baladrón; R Galán; N Bejarano; L Medina-Prado; N Villaseca; J M Pérez-Ortiz; J R Muñoz-Rodríguez; J L Santiago; F J Redondo
Journal:  Rev Esp Quimioter       Date:  2019-12-03       Impact factor: 1.553

  4 in total

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