Elizabeth Gerard1, Jennifer A Frontera, Clinton B Wright. 1. Division of Stroke and Critical Care Neurology, Neurological Institute, College of Physicians and Surgeons, Columbia University, 710 West 168th Street, New York, NY 10032, USA.
Abstract
INTRODUCTION: Cerebral arterial vasospasm following aneurysmal subarachnoid hemorrhage (SAH) is an important cause of delayed neurologic deterioration. Vasospasm following isolated intraventricular hemorrhage (IVH) is less common. Accepted predictors of vasospasm following SAH include poor Hunt-Hess grade, elevated transcranial Doppler velocities, and the thickness of cisternal blood on neuroimaging [1, 2]. The role of intraventricular hemorrhage in vasospasm is more controversial. METHODS: Case report and review of the literature. RESULTS: A 41-year-old woman developed symptomatic delayed vasospasm 10 days following isolated IVH due to the rupture of an arteriovenous malformation (AVM). CONCLUSION: Intraventricular hemorrhage can independently cause significant delayed vasospasm. Possible mechanisms are described.
INTRODUCTION:Cerebral arterial vasospasm following aneurysmal subarachnoid hemorrhage (SAH) is an important cause of delayed neurologic deterioration. Vasospasm following isolated intraventricular hemorrhage (IVH) is less common. Accepted predictors of vasospasm following SAH include poor Hunt-Hess grade, elevated transcranial Doppler velocities, and the thickness of cisternal blood on neuroimaging [1, 2]. The role of intraventricular hemorrhage in vasospasm is more controversial. METHODS: Case report and review of the literature. RESULTS: A 41-year-old woman developed symptomatic delayed vasospasm 10 days following isolated IVH due to the rupture of an arteriovenous malformation (AVM). CONCLUSION: Intraventricular hemorrhage can independently cause significant delayed vasospasm. Possible mechanisms are described.
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