Literature DB >> 17521581

A FAScinating receptor in self-tolerance.

Madhu Ramaswamy1, Richard M Siegel.   

Abstract

In this issue of Immunity,Stranges et al. (2007) ablate expression of the death receptor Fas in T cells, B cells, and DC. Fas deficiency in any of these lineages is sufficient to break self-tolerance.

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Year:  2007        PMID： 17521581     DOI： 10.1016/j.immuni.2007.05.004

Source DB:  PubMed          Journal:  Immunity        ISSN： 1074-7613            Impact factor:   31.745


  5 in total

1.  Systemic autoimmunity and defective Fas ligand secretion in the absence of the Wiskott-Aldrich syndrome protein.

Authors:  Nikolay P Nikolov; Masaki Shimizu; Sophia Cleland; Daniel Bailey; Joseph Aoki; Ted Strom; Pamela L Schwartzberg; Fabio Candotti; Richard M Siegel
Journal:  Blood       Date:  2010-05-10       Impact factor: 22.113

Review 2.  Harnessing programmed cell death as a therapeutic strategy in rheumatic diseases.

Authors:  Madhu Ramaswamy; Min Deng; Richard M Siegel
Journal:  Nat Rev Rheumatol       Date:  2011-02-01       Impact factor: 20.543

Review 3.  Beyond TNF: TNF superfamily cytokines as targets for the treatment of rheumatic diseases.

Authors:  Michael Croft; Richard M Siegel
Journal:  Nat Rev Rheumatol       Date:  2017-03-09       Impact factor: 20.543

4.  FAS mRNA editing in Human Systemic Lupus Erythematosus.

Authors:  Jianming Wu; Fenglong Xie; Kun Qian; Andrew W Gibson; Jeffrey C Edberg; Robert P Kimberly
Journal:  Hum Mutat       Date:  2011-08-11       Impact factor: 4.878

Review 5.  The many roles of FAS receptor signaling in the immune system.

Authors:  Andreas Strasser; Philipp J Jost; Shigekazu Nagata
Journal:  Immunity       Date:  2009-02-20       Impact factor: 31.745
  5 in total

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