Literature DB >> 17519409

Sulfadoxine-pyrimethamine, chlorproguanil-dapsone, or chloroquine for the treatment of Plasmodium vivax malaria in Afghanistan and Pakistan: a randomized controlled trial.

Toby Leslie1, M Ismail Mayan, M Anwar Hasan, M Hanif Safi, Eveline Klinkenberg, Christopher J M Whitty, Mark Rowland.   

Abstract

CONTEXT: In areas where Plasmodium falciparum and Plasmodium vivax coexist and treatments for the 2 species differ, misdiagnosis can lead to poor outcomes in either disease. A unified therapy effective against both species would reduce reliance on species-specific diagnosis, which in many areas is difficult to maintain. The antifolates are an important and affordable antimalarial class to which it is often assumed P vivax malaria is intrinsically resistant.
OBJECTIVE: To test the relative efficacy and safety of 2 antifolate drugs against P vivax malaria and compare each with chloroquine. DESIGN, SETTING, AND PATIENTS: An open-label randomized controlled trial comparing chloroquine, sulfadoxine-pyrimethamine, and chlorproguanil-dapsone for the treatment of P vivax malaria was conducted in eastern Afghanistan and northwestern Pakistan, areas in which P vivax malaria predominates. A total of 20,410 patients older than 3 years were screened; 767 patients (315 in Pakistan and 452 in Afghanistan) with confirmed P vivax malaria were enrolled and followed up daily for 4 days, then weekly for 28 days, between March 2004 and June 2006. MAIN OUTCOME MEASURES: Complete clearance of parasites with no recrudescence by day 14. Secondary outcomes included being parasite-free by day 28, clinical failure, and anemia.
RESULTS: By day 14, only 1 patient in the sulfadoxine-pyrimethamine group had parasites. By day 28, failure rates were found in 2 of 153 patients (1.3%) in the chloroquine group, 5 of 290 patients (1.7%) in the sulfadoxine-pyrimethamine group, and 27 of 272 patients (9.9%) in the chlorproguanil-dapsone group. Chlorproguanil-dapsone was less effective than sulfadoxine-pyrimethamine (adjusted odds ratio [OR], 6.4; 95% confidence interval [CI], 2.4-17.0; P<.001) and chloroquine (adjusted OR, 8.4; 95% CI, 2.0-36.5; P = .004). Chloroquine and sulfadoxine-pyrimethamine were equivalent in efficacy at day 28 (adjusted OR, 1.3; 95% CI, 0.3-7.0; P = .73). Chloroquine cleared gametocytes and asexual parasites more rapidly than sulfadoxine-pyrimethamine or chlorproguanil-dapsone did. All drugs were well tolerated.
CONCLUSIONS: Although chloroquine remains the drug of choice, antifolates are effective against P vivax malaria in South Asia. These drugs may be appropriate for unified treatment where species-specific diagnosis is unavailable, most likely in combination with other drugs. TRIAL REGISTRATION: clinicaltrials.gov Identifier: NCT00158561.

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Year:  2007        PMID: 17519409     DOI: 10.1001/jama.297.20.2201

Source DB:  PubMed          Journal:  JAMA        ISSN: 0098-7484            Impact factor:   56.272


  25 in total

Review 1.  Resistance to therapies for infection by Plasmodium vivax.

Authors:  J Kevin Baird
Journal:  Clin Microbiol Rev       Date:  2009-07       Impact factor: 26.132

Review 2.  Epidemiology and infectivity of Plasmodium falciparum and Plasmodium vivax gametocytes in relation to malaria control and elimination.

Authors:  Teun Bousema; Chris Drakeley
Journal:  Clin Microbiol Rev       Date:  2011-04       Impact factor: 26.132

3.  The impact of phenotypic and genotypic G6PD deficiency on risk of plasmodium vivax infection: a case-control study amongst Afghan refugees in Pakistan.

Authors:  Toby Leslie; Marnie Briceño; Ismail Mayan; Nasir Mohammed; Eveline Klinkenberg; Carol Hopkins Sibley; Christopher J M Whitty; Mark Rowland
Journal:  PLoS Med       Date:  2010-05-25       Impact factor: 11.069

4.  Dihydroartemisinin-piperaquine versus chloroquine to treat vivax malaria in Afghanistan: an open randomized, non-inferiority, trial.

Authors:  Ghulam Rahim Awab; Sasithon Pukrittayakamee; Mallika Imwong; Arjen M Dondorp; Charles J Woodrow; Sue Jean Lee; Nicholas P J Day; Pratap Singhasivanon; Nicholas J White; Faizullah Kaker
Journal:  Malar J       Date:  2010-04-21       Impact factor: 2.979

5.  Prevalence of antimalarial drug resistance mutations in Plasmodium vivax and P. falciparum from a malaria-endemic area of Pakistan.

Authors:  Lubna Khatoon; Frederick N Baliraine; Mariangela Bonizzoni; Salman A Malik; Guiyun Yan
Journal:  Am J Trop Med Hyg       Date:  2009-09       Impact factor: 2.345

6.  Prevalence of polymorphisms in antifolate drug resistance molecular marker genes pvdhfr and pvdhps in clinical isolates of Plasmodium vivax from Kolkata, India.

Authors:  Swagata Ganguly; Pabitra Saha; Moytrey Chatterjee; Ardhendu K Maji
Journal:  Antimicrob Agents Chemother       Date:  2013-10-21       Impact factor: 5.191

Review 7.  The anaemia of Plasmodium vivax malaria.

Authors:  Nicholas M Douglas; Nicholas M Anstey; Pierre A Buffet; Jeanne R Poespoprodjo; Tsin W Yeo; Nicholas J White; Ric N Price
Journal:  Malar J       Date:  2012-04-27       Impact factor: 2.979

8.  Field trial of three different Plasmodium vivax-detecting rapid diagnostic tests with and without evaporative cool box storage in Afghanistan.

Authors:  Amy F W Mikhail; Toby J Leslie; Mohammad I Mayan; Rohullah Zekria; Nader Mohammad; Mohammad A Hasanzai; Najibullah Safi; Christopher J M Whitty; Mark Rowland
Journal:  Malar J       Date:  2011-06-22       Impact factor: 2.979

Review 9.  A systematic review of the reporting of Data Monitoring Committees' roles, interim analysis and early termination in pediatric clinical trials.

Authors:  Ricardo M Fernandes; Johanna H van der Lee; Martin Offringa
Journal:  BMC Pediatr       Date:  2009-12-13       Impact factor: 2.125

10.  In vivo Susceptibility of Plasmodium vivax to Chloroquine in Southeastern Iran.

Authors:  A Heidari; H Keshavarz; S Shojaee; A Raeisi; S Dittrich
Journal:  Iran J Parasitol       Date:  2012       Impact factor: 1.012

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