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15 in total

1. Overexpression of Bcl-2 in the intestinal epithelium improves survival in septic mice.

Authors: Craig M Coopersmith; Katherine C Chang; Paul E Swanson; Kevin W Tinsley; Paul E Stromberg; Timothy G Buchman; Irene E Karl; Richard S Hotchkiss
Journal: Crit Care Med Date: 2002-01 Impact factor: 7.598

Review 2. NF-kappaB activation.

Authors: E Abraham
Journal: Crit Care Med Date: 2000-04 Impact factor: 7.598

3. Modulation of barrier function during Fas-mediated apoptosis in human intestinal epithelial cells.

Authors: M T Abreu; A A Palladino; E T Arnold; R S Kwon; J A McRoberts
Journal: Gastroenterology Date: 2000-12 Impact factor: 22.682

Review 4. The NF-kappaB-mediated control of the JNK cascade in the antagonism of programmed cell death in health and disease.

Authors: S Papa; C Bubici; F Zazzeroni; C G Pham; C Kuntzen; J R Knabb; K Dean; G Franzoso
Journal: Cell Death Differ Date: 2006-05 Impact factor: 15.828

5. The two faces of IKK and NF-kappaB inhibition: prevention of systemic inflammation but increased local injury following intestinal ischemia-reperfusion.

Authors: Lee-Wei Chen; Laurence Egan; Zhi-Wei Li; Florian R Greten; Martin F Kagnoff; Michael Karin
Journal: Nat Med Date: 2003-04-07 Impact factor: 53.440

10. The IKKbeta subunit of IkappaB kinase (IKK) is essential for nuclear factor kappaB activation and prevention of apoptosis.

Authors: Z W Li; W Chu; Y Hu; M Delhase; T Deerinck; M Ellisman; R Johnson; M Karin
Journal: J Exp Med Date: 1999-06-07 Impact factor: 14.307

Just the right amount of JNK: How nuclear factor-kappaB and downstream mediators prevent burn-induced intestinal injury.

1. Overexpression of Bcl-2 in the intestinal epithelium improves survival in septic mice.

Review 2. NF-kappaB activation.

3. Modulation of barrier function during Fas-mediated apoptosis in human intestinal epithelial cells.

Review 4. The NF-kappaB-mediated control of the JNK cascade in the antagonism of programmed cell death in health and disease.

5. The two faces of IKK and NF-kappaB inhibition: prevention of systemic inflammation but increased local injury following intestinal ischemia-reperfusion.

6. The E3 ubiquitin ligase itch couples JNK activation to TNFalpha-induced cell death by inducing c-FLIP(L) turnover.

7. Role of TNF-alpha in gut mucosal changes after severe burn.

8. A JNK-dependent pathway is required for TNFalpha-induced apoptosis.

9. JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species.

10. The IKKbeta subunit of IkappaB kinase (IKK) is essential for nuclear factor kappaB activation and prevention of apoptosis.