Literature DB >> 17400664

World Trade Center "sarcoid-like" granulomatous pulmonary disease in New York City Fire Department rescue workers.

Gabriel Izbicki1, Robert Chavko, Gisela I Banauch, Michael D Weiden, Kenneth I Berger, Thomas K Aldrich, Charles Hall, Kerry J Kelly, David J Prezant.   

Abstract

BACKGROUND: Previous reports suggest that sarcoidosis occurs with abnormally high frequency in firefighters. We sought to determine whether exposure to World Trade Center (WTC) "dust" during the collapse and rescue/recovery effort increased the incidence of sarcoidosis or "sarcoid-like" granulomatous pulmonary disease (SLGPD).
METHODS: During the 5 years after the WTC disaster, enrollees in the Fire Department of New York (FDNY) WTC monitoring and treatment programs who had chest radiograph findings suggestive of sarcoidosis underwent evaluation, including the following: chest CT imaging, pulmonary function, provocative challenge, and biopsy. Annual incidence rates were compared to the 15 years before the WTC disaster.
RESULTS: After WTC dust exposure, pathologic evidence consistent with new-onset sarcoidosis was found in 26 patients: all 26 patients had intrathoracic adenopathy, and 6 patients (23%) had extrathoracic disease. Thirteen patients were identified during the first year after WTC dust exposure (incidence rate, 86/100,000), and 13 patients were identified during the next 4 years (average annual incidence rate, 22/100,000; as compared to 15/100,000 during the 15 years before the WTC disaster). Eighteen of 26 patients (69%) had findings consistent with asthma. Eight of 21 patients (38%) agreeing to challenge testing had airway hyperreactivity (AHR), findings not seen in FDNY sarcoidosis patients before the WTC disaster.
CONCLUSION: After the WTC disaster, the incidence of sarcoidosis or SLGPD was increased among FDNY rescue workers. This new information about the early onset of WTC-SLGPD and its association with asthma/AHR has important public health consequences for disease prevention, early detection, and treatment following environmental/occupational exposures.

Entities:  

Mesh:

Substances:

Year:  2007        PMID: 17400664     DOI: 10.1378/chest.06-2114

Source DB:  PubMed          Journal:  Chest        ISSN: 0012-3692            Impact factor:   9.410


  81 in total

1.  Roles of MAPK pathway activation during cytokine induction in BEAS-2B cells exposed to fine World Trade Center (WTC) dust.

Authors:  Shang Wang; Colette Prophete; Joleen M Soukup; Lung-Chi Chen; Max Costa; Andrew Ghio; Qingshan Qu; Mitchell D Cohen; Haobin Chen
Journal:  J Immunotoxicol       Date:  2010-08-24       Impact factor: 3.000

2.  Emerging exposures and respiratory health: World Trade Center dust.

Authors:  William N Rom; Joan Reibman; Linda Rogers; Michael D Weiden; Beno Oppenheimer; Kenneth Berger; Roberta Goldring; Denise Harrison; David Prezant
Journal:  Proc Am Thorac Soc       Date:  2010-05

Review 3.  Sarcoidosis--scientific progress and clinical challenges.

Authors:  Edward S Chen; David R Moller
Journal:  Nat Rev Rheumatol       Date:  2011-07-12       Impact factor: 20.543

4.  Mental health, long-term medication adherence, and the control of asthma symptoms among persons exposed to the WTC 9/11 disaster.

Authors:  Jennifer Brite; Stephen Friedman; Rafael E de la Hoz; Joan Reibman; James Cone
Journal:  J Asthma       Date:  2019-10-10       Impact factor: 2.515

5.  Obstructive airways disease with air trapping among firefighters exposed to World Trade Center dust.

Authors:  Michael D Weiden; Natalia Ferrier; Anna Nolan; William N Rom; Ashley Comfort; Jackson Gustave; Rachel Zeig-Owens; Shugi Zheng; Roberta M Goldring; Kenneth I Berger; Kaitlyn Cosenza; Roy Lee; Mayris P Webber; Kerry J Kelly; Thomas K Aldrich; David J Prezant
Journal:  Chest       Date:  2009-10-09       Impact factor: 9.410

6.  Histological Diagnoses of Military Personnel Undergoing Lung Biopsy After Deployment to Southwest Asia.

Authors:  Cristian S Madar; Michael R Lewin-Smith; Teri J Franks; Russell A Harley; John S Klaric; Michael J Morris
Journal:  Lung       Date:  2017-05-23       Impact factor: 2.584

7.  Transcriptional survey of alveolar macrophages in a murine model of chronic granulomatous inflammation reveals common themes with human sarcoidosis.

Authors:  Arjun Mohan; Anagha Malur; Matthew McPeek; Barbara P Barna; Lynn M Schnapp; Mary Jane Thomassen; Sina A Gharib
Journal:  Am J Physiol Lung Cell Mol Physiol       Date:  2017-12-06       Impact factor: 5.464

8.  PPAR-gamma pathways attenuate pulmonary granuloma formation in a carbon nanotube induced murine model of sarcoidosis.

Authors:  Matthew McPeek; Anagha Malur; Debra A Tokarz; Gina Murray; Barbara P Barna; Mary Jane Thomassen
Journal:  Biochem Biophys Res Commun       Date:  2018-06-15       Impact factor: 3.575

9.  Case report: Lung disease in World Trade Center responders exposed to dust and smoke: carbon nanotubes found in the lungs of World Trade Center patients and dust samples.

Authors:  Maoxin Wu; Ronald E Gordon; Robin Herbert; Maria Padilla; Jacqueline Moline; David Mendelson; Virginia Litle; William D Travis; Joan Gil
Journal:  Environ Health Perspect       Date:  2010-04       Impact factor: 9.031

Review 10.  Newly recognized occupational and environmental causes of chronic terminal airways and parenchymal lung disease.

Authors:  Maor Sauler; Mridu Gulati
Journal:  Clin Chest Med       Date:  2012-12       Impact factor: 2.878

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.