Literature DB >> 17389802

A chronic progressive case of enteroviral limbic encephalitis associated with autoantibody to glutamate receptor epsilon2.

Hideto Nakajima, Mitsuaki Hosoya, Yukitoshi Takahashi, Kuniko Matsuyama, Muneyoshi Tagami, Simon Ishida, Daisuke Furutama, Masakazu Sugino, Fumiharu Kimura, Kei-ichi Shinoda, Toshiaki Hanafusa.   

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Year:  2007        PMID： 17389802     DOI： 10.1159/000101289

Source DB:  PubMed          Journal:  Eur Neurol        ISSN： 0014-3022            Impact factor:   1.710


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  3 in total

1.  Enterovirus 71-related encephalomyelitis: usual and unusual magnetic resonance imaging findings.

Authors:  Seonah Jang; Sang-il Suh; Su Min Ha; Jung Hye Byeon; Baik-Lin Eun; Young Hen Lee; Hyung Suk Seo; So-Hee Eun; Hae-Young Seol
Journal:  Neuroradiology       Date:  2011-08-24       Impact factor: 2.804

2.  Search for Viral Infections in Cerebrospinal Fluid From Patients With Autoimmune Encephalitis.

Authors:  Karol Perlejewski; Agnieszka Pawełczyk; Iwona Bukowska-Ośko; Małgorzata Rydzanicz; Tomasz Dzieciątkowski; Marcin Paciorek; Michał Makowiecki; Kamila Caraballo Cortés; Marta Grochowska; Marek Radkowski; Tomasz Laskus
Journal:  Open Forum Infect Dis       Date:  2020-10-07       Impact factor: 3.835

Review 3.  Glutamate receptor antibodies in neurological diseases: anti-AMPA-GluR3 antibodies, anti-NMDA-NR1 antibodies, anti-NMDA-NR2A/B antibodies, anti-mGluR1 antibodies or anti-mGluR5 antibodies are present in subpopulations of patients with either: epilepsy, encephalitis, cerebellar ataxia, systemic lupus erythematosus (SLE) and neuropsychiatric SLE, Sjogren's syndrome, schizophrenia, mania or stroke. These autoimmune anti-glutamate receptor antibodies can bind neurons in few brain regions, activate glutamate receptors, decrease glutamate receptor's expression, impair glutamate-induced signaling and function, activate blood brain barrier endothelial cells, kill neurons, damage the brain, induce behavioral/psychiatric/cognitive abnormalities and ataxia in animal models, and can be removed or silenced in some patients by immunotherapy.

Authors:  Mia Levite
Journal:  J Neural Transm (Vienna)       Date:  2014-08-01       Impact factor: 3.575
  3 in total

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