Literature DB >> 17382120

Cardiac manifestations in oxidative phosphorylation disorders of childhood.

J Yaplito-Lee1, R Weintraub, K Jamsen, C W Chow, D R Thorburn, A Boneh.   

Abstract

OBJECTIVE: To determine the frequency, type, and severity of cardiac involvement in pediatric patients with oxidative phosphorylation (OXPHOS) disorders. STUDY
DESIGN: Retrospective review of clinical and laboratory records of all patients with definitive OXPHOS disorders diagnosed and treated at the Royal Children's Hospital in Melbourne between 1984 and 2005.
RESULTS: Of a total of 89 patients (male:female ratio 1.5:1) 29 (33%) had cardiac involvement: 9 as presenting symptoms, 9 developing on follow-up, and 11 with subclinical cardiac findings. Leigh or Leigh-like syndrome and complex I and combined complex I, III, and IV deficiencies were the most common clinical and laboratory diagnoses, respectively. Clinically symptomatic patients had hypertrophic cardiomyopathy (5 patients), dilated cardiomyopathy (4 patients), combined ventricular hypertrophy and systolic dysfunction (3 patients), and left ventricular noncompaction (3 patients) at first assessment. A change in the type of cardiomyopathy was noted on follow-up in 2 patients. Conduction and rhythm abnormalities were present in 7 symptomatic patients.
CONCLUSIONS: Cardiac assessment in children with OXPHOS disorders may reveal subclinical abnormalities of cardiac function. Patients who present with primary cardiac features have a poor prognosis. OXPHOS disorders should be considered in the differential diagnosis of children presenting with otherwise unexplained cardiomyopathy.

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Year:  2007        PMID: 17382120     DOI: 10.1016/j.jpeds.2006.12.047

Source DB:  PubMed          Journal:  J Pediatr        ISSN: 0022-3476            Impact factor:   4.406


  18 in total

Review 1.  Mitochondrial Diseases Part I: mouse models of OXPHOS deficiencies caused by defects in respiratory complex subunits or assembly factors.

Authors:  Alessandra Torraco; Susana Peralta; Luisa Iommarini; Francisca Diaz
Journal:  Mitochondrion       Date:  2015-02-04       Impact factor: 4.160

2.  Implications of genetic testing in noncompaction/hypertrabeculation.

Authors:  Joseph T C Shieh
Journal:  Am J Med Genet C Semin Med Genet       Date:  2013-07-10       Impact factor: 3.908

3.  Tissue-specific splicing of an Ndufs6 gene-trap insertion generates a mitochondrial complex I deficiency-specific cardiomyopathy.

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Journal:  Proc Natl Acad Sci U S A       Date:  2012-04-02       Impact factor: 11.205

Review 4.  Cardiovascular autonomic neuropathies as complications of diabetes mellitus.

Authors:  Michael Kuehl; Martin J Stevens
Journal:  Nat Rev Endocrinol       Date:  2012-02-28       Impact factor: 43.330

Review 5.  The Genetic Challenges and Opportunities in Advanced Heart Failure.

Authors:  Fady Hannah-Shmouni; Sara B Seidelmann; Sandra Sirrs; Arya Mani; Daniel Jacoby
Journal:  Can J Cardiol       Date:  2015-08-21       Impact factor: 5.223

6.  Mitochondrial Trifunctional Protein Deficiency: Severe Cardiomyopathy and Cardiac Transplantation.

Authors:  C Bursle; R Weintraub; C Ward; R Justo; J Cardinal; D Coman
Journal:  JIMD Rep       Date:  2017-11-10

7.  Exome sequencing identifies mitochondrial alanyl-tRNA synthetase mutations in infantile mitochondrial cardiomyopathy.

Authors:  Alexandra Götz; Henna Tyynismaa; Liliya Euro; Pekka Ellonen; Tuulia Hyötyläinen; Tiina Ojala; Riikka H Hämäläinen; Johanna Tommiska; Taneli Raivio; Matej Oresic; Riitta Karikoski; Outi Tammela; Kalle O J Simola; Anders Paetau; Tiina Tyni; Anu Suomalainen
Journal:  Am J Hum Genet       Date:  2011-05-05       Impact factor: 11.025

Review 8.  Cardiogenetics, neurogenetics, and pathogenetics of left ventricular hypertrabeculation/noncompaction.

Authors:  Josef Finsterer
Journal:  Pediatr Cardiol       Date:  2009-01-29       Impact factor: 1.655

Review 9.  Cell-permeable protein therapy for complex I dysfunction.

Authors:  Salvatore Pepe; Robert M Mentzer; Roberta A Gottlieb
Journal:  J Bioenerg Biomembr       Date:  2014-07-09       Impact factor: 2.945

Review 10.  Mitochondrial disease in childhood: nuclear encoded.

Authors:  Amy C Goldstein; Poonam Bhatia; Jodie M Vento
Journal:  Neurotherapeutics       Date:  2013-04       Impact factor: 7.620

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