Literature DB >> 17346749

Evidence for astrocytosis in ALS demonstrated by [11C](L)-deprenyl-D2 PET.

Anders Johansson1, Henry Engler, Gunnar Blomquist, Berit Scott, Anders Wall, Sten-Magnus Aquilonius, Bengt Långström, Håkan Askmark.   

Abstract

OBJECTIVE: To use deuterium-substituted [11C](L)-deprenyl PET to depict astrocytosis in vivo in patients with amyotrophic lateral sclerosis (ALS).
BACKGROUND: In human brain, the enzyme MAO-B is primarily located in astrocytes. L-deprenyl binds to MAO-B and autoradiography with 3H-L-deprenyl has been used to map astrocytosis in vitro. Motor neuron loss in ALS is accompanied by astrocytosis and astrocytes may play an active role in the neurodegenerative process. Deuterium-substituted [11C](L)-deprenyl PET provides an opportunity to localize astrocytosis in vivo in the brain of patients with ALS.
METHODS: Deuterium-substituted [11C](L)-deprenyl PET was performed in seven patients with ALS and seven healthy control subjects.
RESULTS: Increased uptake rate of [11C](L)-deprenyl was demonstrated in ALS in pons and white matter.
CONCLUSION: This study provides evidence that astrocytosis may be detected in vivo in ALS by the use of deuterium-substituted [11C](L)-deprenyl PET though further studies are needed to determine whether deuterium-substituted [11C](L)-deprenyl binding tracks disease progression and reflects astrocytosis.

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Year:  2007        PMID: 17346749     DOI: 10.1016/j.jns.2007.01.057

Source DB:  PubMed          Journal:  J Neurol Sci        ISSN: 0022-510X            Impact factor:   3.181


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