Literature DB >> 17293682

The inflammatory response to ischemic acute kidney injury: a result of the 'right stuff' in the 'wrong place'?

Christopher Y Lu1, John Hartono, Martin Senitko, Jianlin Chen.   

Abstract

PURPOSE OF REVIEW: Ischemic acute kidney injury may be exacerbated by an inflammatory response. How injury elicits inflammation remains a major question in understanding acute kidney injury. The present review examines the hypothesis that molecules released by injured cells elicit inflammation. RECENT
FINDINGS: After necrotic death, intracellular molecules find their way into the extracellular space. These molecules include heat shock proteins and HMGB1. Receptors for these proteins include TLR4, TLR2, CD91 and RAGE. These proinflammatory mechanisms may be so useful that nature has evolved mechanisms for programming necrotic death via poly(ADP-ribose) polymerase and cyclophilin D. In addition, apoptosis may also elicit inflammation.
SUMMARY: The concepts discussed in this review are important for clinical medicine. Drugs and genetic manipulation may ameliorate ischemic kidney injury by regulating the inflammatory response to cell injury.

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Year:  2007        PMID: 17293682     DOI: 10.1097/MNH.0b013e3280403c4e

Source DB:  PubMed          Journal:  Curr Opin Nephrol Hypertens        ISSN: 1062-4821            Impact factor:   2.894


  26 in total

1.  Toll-like receptor 4 regulates early endothelial activation during ischemic acute kidney injury.

Authors:  Jianlin Chen; Reji John; James A Richardson; John M Shelton; Xin J Zhou; Yanxia Wang; Qing Qing Wu; John R Hartono; Pamela D Winterberg; Christopher Y Lu
Journal:  Kidney Int       Date:  2010-10-06       Impact factor: 10.612

2.  Early interleukin 6 production by leukocytes during ischemic acute kidney injury is regulated by TLR4.

Authors:  Jianlin Chen; John R Hartono; Reji John; Michael Bennett; Xin Jin Zhou; Yanxia Wang; Qingqing Wu; Pamela D Winterberg; Glenn T Nagami; Christopher Y Lu
Journal:  Kidney Int       Date:  2011-06-01       Impact factor: 10.612

Review 3.  Cell therapy, advanced materials, and new approaches to acute kidney injury.

Authors:  Alexander S Yevzlin; H David Humes
Journal:  Hosp Pract (1995)       Date:  2009-12

4.  Catalpol protects mice against renal ischemia/reperfusion injury via suppressing PI3K/Akt-eNOS signaling and inflammation.

Authors:  Jili Zhu; Xinghua Chen; Huiming Wang; Qi Yan
Journal:  Int J Clin Exp Med       Date:  2015-02-15

Review 5.  The cell secretome, a mediator of cell-to-cell communication.

Authors:  Joseph Zullo; Kei Matsumoto; Sandhya Xavier; Brian Ratliff; Michael S Goligorsky
Journal:  Prostaglandins Other Lipid Mediat       Date:  2015-04-29       Impact factor: 3.072

Review 6.  Oxidant Mechanisms in Renal Injury and Disease.

Authors:  Brian B Ratliff; Wasan Abdulmahdi; Rahul Pawar; Michael S Wolin
Journal:  Antioxid Redox Signal       Date:  2016-04-26       Impact factor: 8.401

Review 7.  Apoptosis and acute kidney injury.

Authors:  Andrea Havasi; Steven C Borkan
Journal:  Kidney Int       Date:  2011-05-11       Impact factor: 10.612

Review 8.  Cell-based strategies for the treatment of kidney dysfunction: a review.

Authors:  Christopher J Pino; Alexander S Yevzlin; James Tumlin; H David Humes
Journal:  Blood Purif       Date:  2012-10-24       Impact factor: 2.614

9.  IRF-1 promotes inflammation early after ischemic acute kidney injury.

Authors:  Yanxia Wang; Reji John; Jianlin Chen; James A Richardson; John M Shelton; Michael Bennett; Xin J Zhou; Glenn T Nagami; Ying Zhang; Qing Qing Wu; Christopher Y Lu
Journal:  J Am Soc Nephrol       Date:  2009-05-14       Impact factor: 10.121

10.  It's all in the family: multiple Toll-like receptors offer promise as novel therapeutic targets for stroke neuroprotection.

Authors:  Philberta Y Leung; Amy Eb Packard; Mary P Stenzel-Poore
Journal:  Future Neurol       Date:  2009
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