Literature DB >> 17262213

[1-(11)C]acetate uptake is not increased in renal cell carcinoma.

J Kotzerke1, C Linné, M Meinhardt, J Steinbach, M Wirth, G Baretton, N Abolmaali, B Beuthien-Baumann.   

Abstract

PURPOSE: The purpose of this study was to investigate the potential of [1-(11)C]acetate (AC) as a metabolic tracer for renal cell cancer in human subjects.
METHODS: Twenty-one patients with suspected kidney tumours were investigated with AC and dynamic PET. AC uptake was scored on a five-step scale. Tumour localisation was known from CT/MRI. Histology was available in 18/21 patients. The results in these 18 patients are reported.
RESULTS: AC uptake by the tumour was less than (n=11), equal to (n=5) or higher than (n=2) uptake in the surrounding renal parenchyma. Histological tumour types showed a typical distribution, with a predominance of clear cell carcinomas (n=14) and only a small number of papillary cell carcinomas (n=2) and oncocytomas (n=2). Only the benign oncocytomas were highly positive with AC.
CONCLUSION: In most kidney tumours the AC accumulation was not higher than in normal kidney parenchyma. Therefore, AC PET cannot be recommend for the characterisation of a renal mass.

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Year:  2007        PMID: 17262213     DOI: 10.1007/s00259-006-0362-5

Source DB:  PubMed          Journal:  Eur J Nucl Med Mol Imaging        ISSN: 1619-7070            Impact factor:   9.236


  12 in total

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