Literature DB >> 17259242

Evaluation of the risk for Tay-Sachs disease in individuals of French Canadian ancestry living in new England.

Dianna C Martin1, Brian L Mark, Barbara L Triggs-Raine, Marvin R Natowicz.   

Abstract

BACKGROUND: The assessment of risk for Tay-Sachs disease (TSD) in individuals of French Canadian background living in New England is an important health issue. In preliminary studies of the enzyme-defined carrier frequency for TSD among Franco-Americans in New England, we found frequencies (1:53) higher than predicted from the incidence of infantile TSD in this region. We have now further evaluated the risk for TSD in the Franco-American population of New England.
METHODS: Using a fluorescence-based assay for beta-hexosaminidase activity, we determined the carrier frequencies for TSD in 2783 Franco-Americans. DNA analysis was used to identify mutations causing enzyme deficiency in TSD carriers.
RESULTS: We determined the enzyme-defined carrier frequency for TSD as 1:65 (95% confidence interval 1:49 to 1:90). DNA-based analysis of 24 of the enzyme-defined carriers revealed 21 with sequence changes: 9 disease-causing, 4 benign, and 8 of unknown significance. Six of the unknowns were identified as c.748G>A p.G250S, a mutation we show by expression analysis to behave similarly to the previously described c.805G>A p.G269S adult-onset TSD mutation. This putative adult-onset TSD c.748G>A p.G250S mutation has a population frequency similar to the common 7.6 kb deletion mutation that occurs in persons of French Canadian ancestry.
CONCLUSIONS: We estimate the frequency of deleterious TSD alleles in Franco-Americans to be 1:73 (95% confidence interval 1:55 to 1:107). These data provide a more complete data base from which to formulate policy recommendations regarding TSD heterozygosity screening in individuals of French Canadian background.

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Year:  2007        PMID: 17259242     DOI: 10.1373/clinchem.2006.082727

Source DB:  PubMed          Journal:  Clin Chem        ISSN: 0009-9147            Impact factor:   8.327


  4 in total

1.  Cerebellar atrophy on top of motor neuron compromise as indicator of late-onset GM2 gangliosidosis.

Authors:  Hans Thomas Hölzer; Felix Boschann; Julia B Hennermann; Gabriele Hahn; Andreas Hermann; Maja von der Hagen; Victoria Tüngler
Journal:  J Neurol       Date:  2021-03-09       Impact factor: 4.849

2.  Screening for Tay-Sachs disease carriers by full-exon sequencing with novel variant interpretation outperforms enzyme testing in a pan-ethnic cohort.

Authors:  Alana C Cecchi; Elizabeth S Vengoechea; Kristjan E Kaseniit; Melanie W Hardy; Laura A Kiger; Nikita Mehta; Imran S Haque; Krista Moyer; Patricia Z Page; Dale Muzzey; Karen A Grinzaid
Journal:  Mol Genet Genomic Med       Date:  2019-07-10       Impact factor: 2.183

3.  Next-generation DNA sequencing of HEXA: a step in the right direction for carrier screening.

Authors:  Jodi D Hoffman; Valerie Greger; Erin T Strovel; Miriam G Blitzer; Mark A Umbarger; Caleb Kennedy; Brian Bishop; Patrick Saunders; Gregory J Porreca; Jaclyn Schienda; Jocelyn Davie; Stephanie Hallam; Charles Towne
Journal:  Mol Genet Genomic Med       Date:  2013-09-16       Impact factor: 2.183

4.  Tay-Sachs Carrier Screening by Enzyme and Molecular Analyses in the New York City Minority Population.

Authors:  Nikita Mehta; Gabriel A Lazarin; Erica Spiegel; Kathleen Berentsen; Kelly Brennan; Jessica Giordano; Imran S Haque; Ronald Wapner
Journal:  Genet Test Mol Biomarkers       Date:  2016-06-30
  4 in total

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