Literature DB >> 17201122

TRAIL expression up-regulated by interferon-gamma via phosphorylation of STAT1 induces myeloma cell death.

Yoshie Miura1, Takayuki Tsujioka, Yasumitsu Nishimura, Haruko Sakaguchi, Megumi Maeda, Hiroaki Hayashi, Maolong Dong, Fuminori Hyodoh, Ken-Ichiro Yata, Hideho Wada, Takashi Sugihara, Takemi Otsuki.   

Abstract

BACKGROUND: Although the cellular and molecular biological effects of interferon (IFN)-alpha have been well-investigated, the effects of IFN-gamma are less understood.
MATERIALS AND METHODS: Eleven human myeloma cell lines with various myeloma-specific chromosomal translocations and overexpression of oncogenes were cultured with 1000 U/ml of IFN-gamma. In the KMS-20 cells, which showed growth inhibition due to IFN-gamma, trail expression, status of the Janus kinase (JAK)/STAT pathway were analyzed.
RESULTS: KMS-20 cells showed marked up-regulation of trail, activation of STAT1 and TRAIL hyperproduction induced by IFN-gamma.
CONCLUSION: The effects of IFN-gamma on growth inhibition of KMS-20 cells were characterized by activation of the JAK/STAT signalling pathway, particularly STAT1 phosphorylation, enhanced secretion of TRAIL, and auto/paracrine usage of secreted TRAIL to induce apoptotic cell death. From these results, IFN-gamma may be considered one of the drugs to be used in future multidrug chemotherapeutic regimens for myeloma patients.

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Year:  2006        PMID: 17201122

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  12 in total

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