Literature DB >> 17178948

Potential role of abnormal ion transport in the pathogenesis of chronic sinusitis.

Kenji Dejima1, Scott H Randell, M Jackson Stutts, Brent A Senior, Richard C Boucher.   

Abstract

OBJECTIVES: To create well-differentiated cultures of normal and chronic sinusitis paranasal sinus epithelial cells and to compare their electrophysiologic properties.
DESIGN: In vitro investigation using primary sinus epithelial cells, initially cultured on plastic tissue culture dishes. Cells were characterized by means of immunocytochemical analysis and then passaged to air-liquid interface culture conditions. The morphologic features of air-liquid interface cultures were assessed using light and electron microscopy. Epithelial Na(+) channel, Na(+)-K(+)-2Cl(-) cotransporter, cystic fibrosis transmembrane conductance regulator, and Ca(2+)-activated Cl(-) channel function were investigated in Ussing chambers.
SUBJECTS: Specimens were obtained from 15 patients undergoing transsphenoidal pituitary procedures, tumor removal, or trauma repair and from 9 patients with chronic sinusitis.
RESULTS: After culture at an air-liquid interface for 21 days, the epithelium was pseudostratified and contained basal, mucous secretory, and ciliated cells. There were no detectable morphologic differences between normal and chronic sinusitis cells. In cultures of normal cells, median basal short circuit current was 4.7 microA/cm(2), and Na(+) transport, defined as the amiloride hydrochloride-sensitive component, was approximately 20% of the total. Basal and amiloride-sensitive short circuit currents were greater in cultures of chronic sinusitis cells. Basal short circuit currents in both types of cultures were insensitive to the Cl(-) transport inhibitor bumetanide, but all responded to forskolin or uridine triphosphate. After amiloride pretreatment, forskolin and uridine triphosphate responses were greater in chronic sinusitis cells.
CONCLUSIONS: We established methods for well-differentiated sinus epithelial cultures. The cells exhibited Na(+) absorption and Cl(-) secretion, and elevated rates of ion transport may be pathophysiologically relevant in chronic sinusitis.

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Year:  2006        PMID: 17178948     DOI: 10.1001/archotol.132.12.1352

Source DB:  PubMed          Journal:  Arch Otolaryngol Head Neck Surg        ISSN: 0886-4470


  16 in total

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Review 3.  Acquired cilia dysfunction in chronic rhinosinusitis.

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4.  Increased expression of the epithelial anion transporter pendrin/SLC26A4 in nasal polyps of patients with chronic rhinosinusitis.

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5.  Characteristics of chloride transport in nasal mucosa from patients with primary ciliary dyskinesia.

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Review 6.  Etiology of epithelial barrier dysfunction in patients with type 2 inflammatory diseases.

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8.  Neutrophils are a major source of the epithelial barrier disrupting cytokine oncostatin M in patients with mucosal airways disease.

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Review 9.  Perspectives on the etiology of chronic rhinosinusitis.

Authors:  Bruce K Tan; Robert P Schleimer; Robert C Kern
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10.  Effect of L-ascorbate on chloride transport in freshly excised sinonasal epithelia.

Authors:  Do-Yeon Cho; Peter H Hwang; Beate Illek
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