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Literature DB >> 17156766

Endothelial nitric oxide synthase uncoupling: Is it a physiological mechanism of endothelium-dependent relaxation in cerebral artery?

Mitsuhiro Yokoyama, Ken-ichi Hirata.

Abstract

Entities: Gene

Mesh：

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Year: 2006 PMID： 17156766 DOI： 10.1016/j.cardiores.2006.11.009

Source DB: PubMed Journal: Cardiovasc Res ISSN： 0008-6363 Impact factor: 10.787

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10 in total

1. Insights into the molecular mechanisms of diabetes-induced endothelial dysfunction: focus on oxidative stress and endothelial progenitor cells.

Authors: Mohamed I Saad; Taha M Abdelkhalek; Moustafa M Saleh; Maher A Kamel; Mina Youssef; Shady H Tawfik; Helena Dominguez
Journal: Endocrine Date: 2015-08-14 Impact factor: 3.633

Review 2. Role of the CYP4A/20-HETE pathway in vascular dysfunction of the Dahl salt-sensitive rat.

Authors: Kathleen M Lukaszewicz; Julian H Lombard
Journal: Clin Sci (Lond) Date: 2013-06 Impact factor: 6.124

Review 3. Molecular Basis of Nitrative Stress in the Pathogenesis of Pulmonary Hypertension.

Authors: Colin E Evans; You-Yang Zhao
Journal: Adv Exp Med Biol Date: 2017 Impact factor: 2.622

4. Hyper-activation of pp60^Src limits nitric oxide signaling by increasing asymmetric dimethylarginine levels during acute lung injury.

Authors: Sanjiv Kumar; Xutong Sun; Satish Kumar Noonepalle; Qing Lu; Evgeny Zemskov; Ting Wang; Saurabh Aggarwal; Christine Gross; Shruti Sharma; Ankit A Desai; Yali Hou; Sridevi Dasarathy; Ning Qu; Vijay Reddy; Sung Gon Lee; Mary Cherian-Shaw; Jason X-J Yuan; John D Catravas; Ruslan Rafikov; Joe G N Garcia; Stephen M Black
Journal: Free Radic Biol Med Date: 2016-11-09 Impact factor: 7.376

Endothelial nitric oxide synthase uncoupling: Is it a physiological mechanism of endothelium-dependent relaxation in cerebral artery?

1. Insights into the molecular mechanisms of diabetes-induced endothelial dysfunction: focus on oxidative stress and endothelial progenitor cells.

Review 2. Role of the CYP4A/20-HETE pathway in vascular dysfunction of the Dahl salt-sensitive rat.

Review 3. Molecular Basis of Nitrative Stress in the Pathogenesis of Pulmonary Hypertension.

4. Hyper-activation of pp60^Src limits nitric oxide signaling by increasing asymmetric dimethylarginine levels during acute lung injury.

Review 5. Oxidative stress in early diabetic nephropathy: fueling the fire.

6. Characteristics and function of cardiac mitochondrial nitric oxide synthase.

Review 7. Reactive oxygen species in pulmonary vascular remodeling.

8. Aldose reductase mediates endothelial cell dysfunction induced by high uric acid concentrations.

9. Oral delivery of xenon for cardiovascular protection.

10. A reply to 'Metabolic effects of sapropterin treatment in autism spectrum disorder: a preliminary study'.

Endothelial nitric oxide synthase uncoupling: Is it a physiological mechanism of endothelium-dependent relaxation in cerebral artery?

1. Insights into the molecular mechanisms of diabetes-induced endothelial dysfunction: focus on oxidative stress and endothelial progenitor cells.

Review 2. Role of the CYP4A/20-HETE pathway in vascular dysfunction of the Dahl salt-sensitive rat.

Review 3. Molecular Basis of Nitrative Stress in the Pathogenesis of Pulmonary Hypertension.

4. Hyper-activation of pp60Src limits nitric oxide signaling by increasing asymmetric dimethylarginine levels during acute lung injury.

Review 5. Oxidative stress in early diabetic nephropathy: fueling the fire.

6. Characteristics and function of cardiac mitochondrial nitric oxide synthase.

Review 7. Reactive oxygen species in pulmonary vascular remodeling.

8. Aldose reductase mediates endothelial cell dysfunction induced by high uric acid concentrations.

9. Oral delivery of xenon for cardiovascular protection.

10. A reply to 'Metabolic effects of sapropterin treatment in autism spectrum disorder: a preliminary study'.

4. Hyper-activation of pp60^Src limits nitric oxide signaling by increasing asymmetric dimethylarginine levels during acute lung injury.