Literature DB >> 17117947

Investigation of the functional variant c.-169T > C of the Fc receptor-like 3 (FCRL3) gene in alopecia areata.

N Schäfer1, B Blaumeiser, T Becker, Y Freudenberg-Hua, S Hanneken, S Eigelshoven, C Schmael, J Lambert, J De Weert, R Kruse, M M Nöthen, R C Betz.   

Abstract

A functional variant in the Fc receptor-like 3 (FCRL3) gene has been implicated in susceptibility to autoimmune diseases such as rheumatoid arthritis, systemic lupus erythematosus and autoimmune thyroid disease. Investigating a large case-control sample of patients with alopecia areata (AA), we found no evidence for the involvement of FCRL3 in susceptibility to AA.

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Year:  2006        PMID: 17117947     DOI: 10.1111/j.1744-313X.2006.00633.x

Source DB:  PubMed          Journal:  Int J Immunogenet        ISSN: 1744-3121            Impact factor:   1.466


  2 in total

1.  Association between Fc receptor-like 3 C169T polymorphism and risk of systemic lupus erythematosus: a meta-analysis.

Authors:  Chen Mao; Haifeng Pan; Qing Chen; Xiwen Wang; Dongqing Ye; Lixin Qiu
Journal:  Mol Biol Rep       Date:  2009-06-30       Impact factor: 2.316

2.  Fc receptor-like 3 (FCRL3) -169 C/T polymorphism and systemic lupus erythematosus: a meta-analysis.

Authors:  Gwan Gyu Song; Jae-Hoon Kim; Sung Jae Choi; Jong Dae Ji; Young Ho Lee
Journal:  Rheumatol Int       Date:  2013-03-20       Impact factor: 2.631

  2 in total

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