Literature DB >> 17099510

The effect of burn injury on CD8+ and CD4+ T cells in an irradiation model of homeostatic proliferation.

Ian B Buchanan1, Robert Maile, Jeffrey A Frelinger, Jeffrey H Fair, Anthony A Meyer, Bruce A Cairns.   

Abstract

BACKGROUND: Homeostatic proliferation of T cells has recently been shown to be an important mechanism in the host response to infection. However, its role in the T cell response to burn injury is unknown. In this study, we examine the effect of burn injury on CD4+ and CD8+ T cell homeostatic proliferation after irradiation.
METHODS: Wild-type C57BL/6 female mice were irradiated with six grays ionizing radiation and 48 hours later, syngeneic whole splenocytes or purified CD4+ or CD8+ T cells labeled with carboxy-fluorescein diacetate, succinimidyl ester were adoptively transferred. Two days later, mice underwent a 20% burn injury, followed by splenocyte harvest 3 and 10 days after injury.
RESULTS: Burn mice demonstrate increased splenic cellularity and CD8+ T cell proliferation after adoptive transfer of either purified CD8+ cells or whole spleen populations compared with unburned (sham) mice. In contrast, CD4+ T cell proliferation after burn injury is unchanged after adoptive transfer of whole spleen cells and drastically decreased after adoptive transfer of a purified CD4+ population compared with sham mice. Ten days after burn injury CD8+ T cells continue to demonstrate greater proliferation than CD4+ T cells.
CONCLUSIONS: CD8+ T cells are more robust than CD4+ T cells in their proliferative response after burn injury. In addition, CD8+ T cell proliferation appears less reliant on other immune cells than purified CD4+ T cell proliferation. These data reiterate the importance of CD8+ T cells in the initial immune response to burn injury.

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Year:  2006        PMID: 17099510     DOI: 10.1097/01.ta.0000195984.56153.21

Source DB:  PubMed          Journal:  J Trauma        ISSN: 0022-5282


  8 in total

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Authors:  Samantha M Valvis; Jason Waithman; Fiona M Wood; Mark W Fear; Vanessa S Fear
Journal:  J Invest Dermatol       Date:  2015-05-07       Impact factor: 8.551

2.  Blocking CXCL1-dependent neutrophil recruitment prevents immune damage and reduces pulmonary bacterial infection after inhalation injury.

Authors:  Julia L M Dunn; Laurel B Kartchner; Wesley H Stepp; Lindsey I Glenn; Madison M Malfitano; Samuel W Jones; Claire M Doerschuk; Robert Maile; Bruce A Cairns
Journal:  Am J Physiol Lung Cell Mol Physiol       Date:  2018-01-25       Impact factor: 5.464

3.  LED phototherapy in full-thickness burns induced by CO2 laser in rats skin.

Authors:  Milene da Silva Melo; Leandro Procópio Alves; Adriana Barrinha Fernandes; Henrique Cunha Carvalho; Carlos José de Lima; Egberto Munin; Mônica Fernandes Gomes; Miguel Angel Castillo Salgado; Renato Amaro Zângaro
Journal:  Lasers Med Sci       Date:  2018-04-27       Impact factor: 3.161

4.  Effect of major burns on early and late activating markers of peripheral blood T lymphocytes.

Authors:  S Sayed; R Bakry; M El-Shazly; M El-Oteify; S Terzaki; M Fekry
Journal:  Ann Burns Fire Disasters       Date:  2012-03-31

5.  Mammalian target of rapamycin regulates a hyperresponsive state in pulmonary neutrophils late after burn injury.

Authors:  Julia L M Dunn; Laurel B Kartchner; Karli Gast; Marci Sessions; Rebecca A Hunter; Lance Thurlow; Anthony Richardson; Mark Schoenfisch; Bruce A Cairns; Robert Maile
Journal:  J Leukoc Biol       Date:  2018-02-02       Impact factor: 4.962

6.  Radiation combined with thermal injury induces immature myeloid cells.

Authors:  April Elizabeth Mendoza; Crystal Judith Neely; Anthony G Charles; Laurel Briane Kartchner; Willie June Brickey; Amal Lina Khoury; Gregory D Sempowski; Jenny P Y Ting; Bruce A Cairns; Robert Maile
Journal:  Shock       Date:  2012-11       Impact factor: 3.454

7.  Burn injury induces high levels of phosphorylated insulin-like growth factor binding protein-1.

Authors:  April E Mendoza; Laura A Maile; Bruce A Cairns; Robert Maile
Journal:  Int J Burns Trauma       Date:  2013-11-01

8.  Th17 (IFNγ- IL17+) CD4+ T cells generated after burn injury may be a novel cellular mechanism for postburn immunosuppression.

Authors:  Crystal J Neely; Robert Maile; Ming-Jin Wang; Sivaram Vadlamudi; Anthony A Meyer; Bruce A Cairns
Journal:  J Trauma       Date:  2011-03
  8 in total

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